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Related Concept Videos

Coronary Artery Disease III: Clinical Manifestations01:30

Coronary Artery Disease III: Clinical Manifestations

Coronary Artery Disease (CAD) is a primary health risk worldwide, leading to significant morbidity and mortality. The condition arises from the buildup of atherosclerotic plaques within the coronary arteries, resulting in diminished blood supply to the heart muscle.The clinical manifestations of CAD vary widely, from asymptomatic stages to severe, life-threatening conditions. Understanding these manifestations is crucial for early diagnosis and effective management.Angina Pectoris: The Warning...
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Heart failure (HF) manifests primarily as dyspnea, fatigue, and fluid retention, resulting in peripheral and pulmonary edema. Symptoms may vary depending on which ventricle is more affected, left or right.Left-Sided Heart FailureAlso known as left ventricular failure, this condition results from the left ventricle's inability to fill or eject sufficient blood into the systemic circulation. It leads to pulmonary congestion, which occurs when the left ventricle fails to eject blood effectively...
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Angina manifests as chest pain, tightness, or squeezing discomfort typically located behind the breastbone. It can radiate to the neck, jaw, shoulders, and inner aspects of the upper arms, most commonly the left arm. Patients may experience shortness of breath, fatigue, profuse sweating, dizziness, indigestion, heartburn, palpitations, anxiety, and vomiting as accompanying symptoms. This pain often lasts a few minutes and is triggered by physical exertion, emotional stress, heavy meals, or cold...
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Pain serves as a critical warning signal that alerts the body to potential or actual harm. When mechanical pressure on the skin is intense, such as from a sharp pinch, the sensation transitions from touch to pain. Similarly, extreme temperatures, like a hot pot handle, convert the sensation of heat into pain. Pain can also result from overstimulation of other senses, such as blinding light, loud noise, or the intense heat from habañero peppers. This ability to sense pain is essential for...
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3D-Neuronavigation In Vivo Through a Patient's Brain During a Spontaneous Migraine Headache
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When the worst headache becomes the worst heartache!

Abdul Hakeem1, Adam D Marks, Sabha Bhatti

  • 1Department of Medicine, University of Wisconsin Hospital and Clinics, 2705 University Avenue #11, Madison, WI 53705, USA. a.hakeem@hosp.wisc.edu

Stroke
|October 20, 2007
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Summary

Subarachnoid hemorrhage (SAH) can trigger Takotsubo cardiomyopathy, a transient left ventricular apical ballooning syndrome. This condition, characterized by ECG changes and wall motion abnormalities, typically resolves within days with a good prognosis.

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Area of Science:

  • Cardiology
  • Neurology

Background:

  • Subarachnoid hemorrhage (SAH) is linked to cardiac complications, including left ventricular abnormalities and ECG changes.
  • Transient left ventricular apical ballooning syndrome, also known as Takotsubo cardiomyopathy, is infrequently reported in SAH patients.

Observation:

  • A 64-year-old woman with SAH from a ruptured posterior communicating artery aneurysm presented with ECG abnormalities and elevated troponin.
  • Transthoracic echocardiogram revealed apical ballooning with reduced ejection fraction (25-30%).

Findings:

  • The patient experienced a full cardiac recovery within 5 days, with normalized ejection fraction and wall motion.
  • This case highlights SAH as a potential trigger for Takotsubo cardiomyopathy.

Implications:

  • Takotsubo cardiomyopathy should be considered in the differential diagnosis for patients with SAH presenting with cardiac symptoms.
  • Early recognition and management can lead to favorable outcomes, though associated morbidity in SAH patients warrants attention.