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Amanita bisporgera. Ingestion and death from mistaken identity
1Division of Emergency Medicine, Children's Hospitals and Clinics of Minnesota, USA.
Abstract:
A triphasic onset of subacute gastrointestinal toxicity, followed by a false recovery period, and, subsequently, late-onset hepatic failure is pathogenomonic of poisoning with cyclopeptide-containing Amanita mushrooms. This article presents the case report of 7 Minnesota residents who developed symptoms of Amanita poisoning after consuming cooked mushrooms picked at a county park. The article includes a review of the pathophysiology and management of Amanita poisoning.
Insights
Poisoning from cyclopeptide-containing Amanita mushrooms causes gastrointestinal issues, a false recovery, and liver failure. This case report details 7 Minnesota residents poisoned after eating wild mushrooms.
Area of Science:
- Toxicology
- Mycology
- Gastroenterology
Background:
- Cyclopeptide-containing Amanita mushrooms are known for severe poisoning.
- Mushroom poisoning can present with a characteristic triphasic illness.
Observation:
- Seven Minnesota residents consumed cooked wild mushrooms.
- All individuals developed symptoms consistent with Amanita mushroom poisoning.
Findings:
- The poisoning followed a pattern of initial gastrointestinal toxicity, a recovery phase, and subsequent liver failure.
- Case highlights the dangers of consuming unidentified wild mushrooms.
Implications:
- Early recognition and management are crucial for Amanita mushroom poisoning.
- Public awareness regarding wild mushroom foraging and identification is essential.
- This case underscores the need for prompt medical intervention to prevent severe hepatic damage.
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