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Published on: February 29, 2020
[Necrotizing otitis externa in a 5-month-old infant]
Mihály Fücsek1, Agnes Kelemen, Péter Jakabos
1Petz Aladár Megyei Oktató Kórház, Gyermek Fül-, Orr-, Gége Részleg, Gyor. drfucsek@t-online.hu
Insights
A 5-month-old infant recovered from severe bilateral otitis externa and sepsis caused by Pseudomonas aeruginosa. Despite aggressive treatment, reconstructive surgery was needed for auditory canal healing.
Area of Science:
- Otolaryngology
- Pediatric Infectious Diseases
- Surgical Reconstruction
Background:
- Necrotising otitis externa is a severe infection, particularly in infants.
- Pseudomonas aeruginosa sepsis can lead to extensive tissue damage.
- Acute mastoiditis can be a precursor to malignant otitis externa.
Observation:
- A 5-month-old infant presented with necrotising bilateral otitis externa, right-sided acute mastoiditis, and Pseudomonas aeruginosa sepsis.
- Despite mastoidectomy, antibiotics, and local treatment, significant necrosis of the external auditory canals occurred.
- Facial nerve paresis was noted but remained stationary throughout treatment.
Findings:
- Repeated necrectomies were required to manage the extensive necrosis.
- Successful bilateral auditory canal reconstructions were performed after inflammation remission.
- The infant healed with residual symptoms after two months, with no underlying immune deficiency or diabetes identified.
Implications:
- This case highlights the severity of necrotising otitis externa in infants and the challenges in treatment.
- Aggressive surgical and medical management, including reconstruction, is crucial for favorable outcomes.
- Early recognition and intervention are vital to prevent complications like facial nerve palsy.
Abstract:
This report presents the case of a healed 5-month-old infant with necrotising (malignant) bilateral otitis externa from acute mastoiditis on the right side and sepsis caused by Pseudomonas aeruginosa infection. Despite of immediately performed mastoidectomy, targeted antibiotics and intensive local treatment, two third of both external auditory canal's epithelium had shown subcutaneous concentric necrosis and ejection which have been removed with repeated necretomies. After the remission of inflammatory symptoms, successful bilateral auditory canal reconstructions were performed. The observed right peripheral facial paresis at the beginning of disease remained stationary. The patient healed with residual symptoms after 2 months of treatment. Neither immune deficiency, nor diabetes could have been proven.
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