Related Experiment Video

Updated: Jul 10, 2026

Mitigation of Blood Borne Cell Attachment to Metal Implants through CD47-Derived Peptide Immobilization
08:13

Mitigation of Blood Borne Cell Attachment to Metal Implants through CD47-Derived Peptide Immobilization

Published on: December 3, 2020

Macrophage tolerance: CD47-SIRP-alpha-mediated signals matter

Hitoshi Takizawa, Markus G Manz

    Nature Immunology
    |November 21, 2007
    PubMed
    Abstract

    No abstract available in PubMed .

    More Related Videos

    Isolation Protocol of Mouse Monocyte-derived Dendritic Cells and Their Subsequent In Vitro Activation with Tumor Immune Complexes
    11:48

    Isolation Protocol of Mouse Monocyte-derived Dendritic Cells and Their Subsequent In Vitro Activation with Tumor Immune Complexes

    Published on: May 31, 2018

    Drug-Induced Senescence in Liver Cells Promotes M2 Macrophage Polarization: Implications for Tyrosine Kinase Inhibitor-Associated Hepatotoxicity
    09:32

    Drug-Induced Senescence in Liver Cells Promotes M2 Macrophage Polarization: Implications for Tyrosine Kinase Inhibitor-Associated Hepatotoxicity

    Published on: October 17, 2025

    Related Experiment Videos

    Last Updated: Jul 10, 2026

    Mitigation of Blood Borne Cell Attachment to Metal Implants through CD47-Derived Peptide Immobilization
    08:13

    Mitigation of Blood Borne Cell Attachment to Metal Implants through CD47-Derived Peptide Immobilization

    Published on: December 3, 2020

    Isolation Protocol of Mouse Monocyte-derived Dendritic Cells and Their Subsequent In Vitro Activation with Tumor Immune Complexes
    11:48

    Isolation Protocol of Mouse Monocyte-derived Dendritic Cells and Their Subsequent In Vitro Activation with Tumor Immune Complexes

    Published on: May 31, 2018

    Drug-Induced Senescence in Liver Cells Promotes M2 Macrophage Polarization: Implications for Tyrosine Kinase Inhibitor-Associated Hepatotoxicity
    09:32

    Drug-Induced Senescence in Liver Cells Promotes M2 Macrophage Polarization: Implications for Tyrosine Kinase Inhibitor-Associated Hepatotoxicity

    Published on: October 17, 2025

    Related Concept Videos

    Phagocytosis of Apoptotic Cells01:17

    Phagocytosis of Apoptotic Cells

    Cells undergoing apoptosis form apoptotic bodies that must be removed immediately to prevent inflammation, autoimmune diseases, and necrosis. Phagocytosis is carried out by professional phagocytes such as macrophages or  immature dendritic cells. Non-professional phagocytes such as  epithelial cells and fibroblasts also take part in this process; however, they are not as effective as professional phagocytes. 
    Normal cells contain receptors that prevent them from being recognized by phagocytes.

    Articles linked to this work by shared authors, journal, and citation graph.

    A fluorescein-conjugated somatostatin receptor antagonist adapter for chimeric antigen receptor T cell therapy of meningioma.

    Neuro-oncology advances·2026

    Quantifying target antigen-dependent CAR T-cell performance against AML.

    Cancer cell international·2026

    A tumor profiling resource for ovarian cancer: insights into chemotherapy-driven heterogeneity and personalized treatment strategy.

    Nature communications·2026

    Skin-derived G-CSF activates pathological granulopoiesis upon psoriasis.

    EMBO molecular medicine·2026

    Prognostic value of measurable residual disease in high-risk MDS after intensive chemotherapy in HOVON-SAKK studies.

    HemaSphere·2026

    A Bispecific Anti-Fluorescein × Anti-CD3 T-cell Engager in Combination with Fluoresceinated Adaptors Enables Lysis of AML Cells.

    Molecular cancer therapeutics·2026

    Peyer's patch M cells organize an epithelial niche that sustains group 3 innate lymphoid cells and IL-22.

    Nature immunology·2026

    Potent type-specific de novo antibodies complement broadly reactive imprinted antibodies in immune responses to SARS-CoV-2 variants.

    Nature immunology·2026

    Epigenetic modulation of stromal cell states underpins pathological tissue niches in Crohn's disease.

    Nature immunology·2026

    TIF1γ regulates stability of regulatory T cells during inflammation.

    Nature immunology·2026

    Loss of cellular RNA homeostasis contributes to MDA5 activation during virus infection.

    Nature immunology·2026

    A novel CAF population coordinates hyper-suppressive regulatory T cell recruitment and localization in lung cancer.

    Nature immunology·2026

    Sodium bicarbonate attenuates myocardial ischemia-reperfusion injury via activation of the splenic cholinergic anti-inflammatory pathway.

    JTCVS open·2026

    Regulatory T cells in the skin: Origin, function, and plasticity.

    JID innovations : skin science from molecules to population health·2026

    Engineered GLUT1-targeted STING polyproagonists: Redox-triggered activation and enhanced endosomal escape for cancer immunotherapy.

    Asian journal of pharmaceutical sciences·2026

    Bap1-mediated deubiquitination determines immune tolerance and anti-tumor immunity via modulating eTreg cell differentiation and ferroptosis.

    Cell death and differentiation·2026

    Lactate-Induced K370 Lactylation of STING Inhibits STING-TBK1 Signaling and Dampens Anti-Tumor Immunity.

    Advanced science (Weinheim, Baden-Wurttemberg, Germany)·2026

    Biomarkers in Irritable Bowel Syndrome: Bridging Gut-Brain Mechanisms to Precision Care.

    Current gastroenterology reports·2026
    See all related articles
    JoVE
    x logofacebook logolinkedin logoyoutube logo
    ABOUT JoVE
    OverviewLeadershipBlogJoVE Help Center
    AUTHORS
    Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
    LIBRARIANS
    TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
    RESEARCH
    JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
    EDUCATION
    JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
    Terms & Conditions of Use
    Privacy Policy
    Policies
    Jove
    Visualize
    Contact Us