[Acute inferior myocardial infarction masking the J wave syndrome. Based on four observations]
1Servicio de Medicina Intensiva, Hospital General de Ciudad Real, España. jortegacar@gmail.com
Insights
J wave syndrome, characterized by prominent J waves, can be masked by acute myocardial infarction. Early detection of J waves during infarction may indicate underlying J wave syndromes like Brugada syndrome.
Area of Science:
- Cardiology
- Electrophysiology
Background:
- J wave syndrome encompasses early repolarization syndrome, Brugada syndrome, and idiopathic ventricular fibrillation, marked by J waves and ST elevation without structural heart disease.
- Acute coronary syndromes are common causes of ST deviation, but their effect on J wave syndrome ECG manifestations is poorly understood.
Observation:
- This report details four cases of acute inferior ST-elevation myocardial infarction.
- Patients initially presented with J waves (or J point depression) and ST depression in right precordial leads.
- These ECG changes resolved, replaced by prominent J waves (R') and anterior ST elevation, suggesting a coexisting J wave syndrome.
Findings:
- Acute inferior myocardial infarction can obscure J wave syndrome.
- ST depression in right precordial leads during inferior myocardial infarction may mask underlying J wave abnormalities.
- The transient ECG changes observed suggest a dynamic interplay between ischemic injury currents and J wave syndrome pathophysiology.
Implications:
- Early identification of J waves or J point depression during acute myocardial infarction is crucial.
- This may aid in diagnosing underlying J wave syndromes, such as early repolarization syndrome or Brugada syndrome, even when masked.
- Recognizing these ECG patterns can improve risk stratification and management of patients with potentially life-threatening arrhythmias.
Abstract:
The J wave syndrome is characterized by a prominent J wave accompanied by ST-segment elevation in the absence of structural heart disease. It includes the benign early repolarization syndrome, the highly arrhythmogenic Brugada syndrome and idiopathic ventricular fibrillation. Although acute coronary syndromes are one of the leading causes of ST-segment deviation, no clinical reports that specifically describe the modulating effects of an ischemic injury current on the ECG manifestations of the J wave syndrome have been found. This report describes four cases of patients with acute inferior ST-segment elevation myocardial infarction who had J wave (or negative deplacement of the J point) and ST-segment depression in the right precordial leads. Later, these precordial ECG alterations disappeared and were progressively replaced by prominent J (R') waves and anterior ST-segment elevations, suggesting the presence of a J wave syndrome. In conclusion, the J wave syndrome may be obscured by an acute inferior myocardial infarction with concomitant ST-segment depression in the right precordial leads. In such circumstances, early detection of the J wave (or depressed J point) may be used as ECG marker of the early repolarization syndrome or Brugada syndrome.
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