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Published on: June 14, 2015
The relationship between cardiac reactivity in the laboratory and in real life
Derek W Johnston1, Martti T Tuomisto, Geoffrey R Patching
1School of Psychology, College of Life Sciences and Medicine, University of Aberdeen, Scotland. d.johnston@abdn.ac.uk
Insights
Excessive cardiovascular response to stress may increase cardiovascular disease risk. This study found that heart rate reactivity in laboratory settings correlated with real-life stress responses, suggesting a link to disease risk.
Area of Science:
- Cardiology
- Psychophysiology
- Stress Research
Background:
- Elevated cardiovascular response to acute stress is a potential risk factor for cardiovascular disease.
- Current assessment relies on laboratory stressor responses, but real-life correlations are crucial.
Purpose of the Study:
- To investigate the relationship between heart rate (HR) response to laboratory stressors and real-life HR reactivity.
- To determine if laboratory-induced cardiovascular reactivity predicts cardiovascular disease risk in daily life.
Main Methods:
- Measured ambulatory heart rate, activity, and posture over a 7-hour period.
- Assessed HR variation, response to public speaking (real-life stressor), and HR increase during self-reported tense arousal.
Main Results:
- Heart rate increase to laboratory stressors showed inconsistent relation to HR variation.
- Laboratory HR response correlated with real-life measures like public speaking and tense arousal.
Conclusions:
- Increased HR reactivity, particularly when combined with stress exposure, may indicate a cardiovascular disease risk.
- Findings support the link between laboratory-assessed cardiovascular reactivity and real-world stress responses.
Objective:
An excessive cardiovascular response to acute stress is a probable risk factor for cardiovascular (CV) disease. Such reactivity is usually assessed from the CV response to laboratory stressors. However, if it is a risk factor, correlated responses must occur in real life.
Design:
In the present study, we investigated the relationship between the heart rate (HR) response to five laboratory stressors and HR reactivity in the field.
Measures:
HR variation, the response to a real life stressor (public speaking), and the increase in HR with periods of self-reported tense arousal. Ambulatory HR, activity and posture were measured continuously over a 7-hr period.
Results:
The HR increase to laboratory stressors did not relate to HR variation consistently, but it did relate to the other two field measures.
Conclusion:
The results suggested that a tendency to increased HR reactivity may be a risk factor for cardiovascular disease when combined with exposure to stress.
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