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Related Concept Videos

Local Anesthetics: Clinical Application as Epidural Anesthesia01:29

Local Anesthetics: Clinical Application as Epidural Anesthesia

Epidural anesthetics are administered in the fat-filled epidural space, the outermost part of the spinal canal. This technique is commonly employed for pain management and anesthesia during lower abdomen and pelvis surgeries or labor and delivery.
Since epidural anesthetics can be infused through an epidural catheter, all types of drugs, including short-acting ones, can be administered. Chloroprocaine and lidocaine are examples of short and long-duration anesthetics, respectively. Bupivacaine...
Local Anesthetics: Clinical Application as Spinal Anesthesia01:11

Local Anesthetics: Clinical Application as Spinal Anesthesia

Spinal anesthetics are given during lower abdomen and limb surgeries to block sensory and motor neurons. They are administered in the mid to low lumbar regions, primarily acting on the cauda equina's nerve roots. The blockade level depends on the local anesthetic (LA) concentration. Usually, low LA concentrations are sufficient to block sensory fibers, while only high LA concentrations block motor fibers. Other factors like injection volume and speed, the patient's posture, and the drug...

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Non-restraining EEG Radiotelemetry: Epidural and Deep Intracerebral Stereotaxic EEG Electrode Placement
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[Pulseless electric activity during postural change after epidural anesthesia].

Jun Ariyama1, Yoshiko Toyama, Yuji Sugimoto

  • 1Division of Anesthesia, Tonami General Hospital, Tonami, Japan.

Masui. the Japanese Journal of Anesthesiology
|February 19, 2008
PubMed
Summary

A patient experienced pulseless electrical activity (PEA) after epidural anesthesia during a posture change. This rare event, linked to anesthesia and diabetic autonomic neuropathy, was successfully managed with therapeutic hypothermia.

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Area of Science:

  • Anesthesiology
  • Cardiology
  • Neurology

Background:

  • A 79-year-old male with poorly controlled diabetes underwent surgery for a left femoral neck fracture.
  • Epidural anesthesia was administered at the L3-4 level in the right lateral position.

Observation:

  • Shortly after repositioning to supine, the patient lost consciousness with unmeasurable blood pressure.
  • Electrocardiogram (ECG) revealed sinus rhythm, leading to a diagnosis of pulseless electrical activity (PEA).

Findings:

  • The patient's loss of consciousness persisted despite blood pressure restoration with epinephrine.
  • Therapeutic hypothermia (34°C for 48 hours) was initiated following the PEA event.
  • No neurological deficits were observed after the patient recovered from therapeutic hypothermia.

Implications:

  • The case suggests a potential link between epidural anesthesia-induced hypotension and diabetic autonomic neuropathy in precipitating PEA.
  • This highlights the importance of vigilant monitoring during anesthetic procedures in patients with complex comorbidities.
  • Therapeutic hypothermia may be a beneficial intervention for neurological protection following such critical events.