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Synergy study of vancomycin or teicoplanin plus gentamicin against enterococci, Staphylococcus aureus and

V Thamlikitkul1

  • 1Department of Medicine, Faculty of Medicine, Siriraj Hospital, Mahidol University, Bangkok, Thailand.

Insights

Vancomycin plus gentamicin and teicoplanin plus gentamicin showed similar efficacy against gram-positive cocci. Synergy was observed in most enterococcal and S. aureus isolates, but less frequently in coagulase-negative staphylococci.

Area of Science:

  • Microbiology
  • Infectious Diseases
  • Pharmacology

Background:

  • Antibiotic resistance is a growing threat, necessitating evaluation of combination therapies.
  • Vancomycin and teicoplanin are key antibiotics for treating infections caused by Gram-positive cocci.
  • Gentamicin is often used in combination with cell-wall active agents to achieve synergistic killing.

Purpose of the Study:

  • To compare the in vitro bactericidal synergy of vancomycin plus gentamicin versus teicoplanin plus gentamicin.
  • To evaluate the efficacy of these combinations against clinical isolates of enterococci, Staphylococcus aureus, and coagulase-negative staphylococci.

Main Methods:

  • A time-kill study design was employed.
  • Bactericidal synergy was defined as a ≥2 log10 decline in colony-forming units (cfu)/mL compared to monotherapy at 24 hours.
  • Antibiotic concentrations were set relative to the minimum inhibitory concentration (MIC) for each isolate.

Main Results:

  • No significant difference in efficacy was observed between vancomycin-gentamicin and teicoplanin-gentamicin combinations.
  • Synergy was demonstrated against 75% of enterococcal, 70% of S. aureus, and 50% of coagulase-negative staphylococcal isolates.
  • Enterococcal isolates resistant to synergy had high gentamicin MICs (>64 mg/L).

Conclusions:

  • Both vancomycin-gentamicin and teicoplanin-gentamicin combinations are effective against a majority of enterococcal and S. aureus isolates.
  • The synergistic effect against staphylococcal isolates was not associated with gentamicin susceptibility, methicillin resistance, or tolerance.
  • High gentamicin MICs may predict resistance to synergistic killing in enterococci.

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