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Transforming growth factor-alpha and the ontogeny of epidermal growth factor receptors in rat kidney

P R Goodyer1, J Fata, C G Goodyer

  • 1Department of Pediatrics, McGill University, Montreal Children's Hospital, Quebec, Canada.

Growth Regulation
|September 1, 1991
PubMed

Insights

Epidermal growth factor (EGF) receptors surge in developing rat kidneys, coinciding with rapid growth. Their decline post-birth correlates with slowed DNA replication, suggesting a role in regulating kidney development.

Area of Science:

  • Developmental Biology
  • Cell Signaling
  • Molecular Endocrinology

Background:

  • Epidermal growth factor (EGF) is known for its mitogenic effects on various cells in vitro.
  • Its precise role in the normal development of organs, particularly the kidney, remains incompletely understood.

Purpose of the Study:

  • To investigate the expression and potential role of epidermal growth factor (EGF) receptors and their ligands during rat kidney development.
  • To correlate EGF receptor dynamics with kidney growth and DNA replication rates.

Main Methods:

  • Quantification of high-affinity EGF/TGF-Alpha receptors using radioligand binding assays at different gestational and post-natal stages.
  • Analysis of Epidermal Growth Factor (EGF) mRNA levels via Northern analysis.
  • Measurement of transforming growth factor-alpha (TGF-α) levels in fetal kidney and amniotic fluid using radioimmunoassay.

Main Results:

  • Specific high-affinity EGF receptors significantly increase in late-gestation rat kidneys, peaking at term, and then decrease post-natally.
  • The decrease in EGF receptor binding post-birth temporally aligns with a slowdown in rat kidney DNA replication.
  • EGF mRNA is undetectable until the second week post-birth, while high levels of TGF-α are present in fetal kidney and amniotic fluid.

Conclusions:

  • EGF receptor induction in the fetal rat kidney may mediate the effects of local TGF-α.
  • These interactions are hypothesized to be critical in dictating the rate of hyperplastic renal growth during the perinatal period.

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