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Fluctuation of fetal rat hepatic histidine decarboxylase activity through the glucocorticoid-ACTH system
E Ohmori1, N Imanishi, M Ohgoh
1Department of Physiological Chemistry, Faculty of Pharmaceutical Sciences, Kyoto University, Japan.
Biochemical Pharmacology
|March 1, 1991
Abstract:
Our studies suggest that the fluctuation of HDC activity in fetal liver in late gestation is regulated by the plasma glucocorticoid level through the pituitary-adrenal system. Taken together, these results support the conclusion that glucocorticoid promotes a rapid increase in HDC synthesis in fetal liver histamine-forming cells, as well as in mouse mastocytoma P-815 cells and rat glandular stomachs.