Sequence and TnphoA analysis of a Mycoplasma hyorhinis protein with membrane export function
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D Yogev1, R Watson-McKown, M A McIntosh
1Department of Molecular Microbiology and Immunology, School of Medicine, University of Missouri, Columbia 65212.
Abstract:
Proteins translocated across the single plasma membrane of mycoplasmas (class Mollicutes) represent important components likely to affect several interactions of these wall-less microbes with their respective hosts. However, identification and functional analysis of such proteins is hampered by the lack of mutational systems in mycoplasmas and by a perceived limitation in translating recombinant mycoplasma genes containing UGA (Trp) codons in other eubacteria. Here we directly analyze a gene encoding a Mycoplasma hyorhinis protein capable of promoting its membrane translocation. It was initially detected by screening a recombinant phage genomic library with antibody from a host with M. hyorhinis-induced arthritis and was localized by Tn5 and deletion mutations affecting expression of antigenic translational products. Sequence analysis of the isolated gene predicted a hydrophilic protein, P101, containing three UGA codons and a putative signal peptide with an uncharacteristic cluster of positively charged amino acids near its C terminus. Nevertheless, lambda::TnphoA transposon mutagenesis of an Escherichia coli plasmid bearing the p101 gene resulted in p101::TnphoA fusions expressing products that could translocate as much as 48 kDa of the P101 sequence (up to the first UGA codon) across the E. coli plasma membrane. Fusion proteins containing mature P101 sequences expressed mycoplasma epitopes and were found by cell fractionation and detergent phase partitioning to be integral membrane proteins in E. coli, suggesting a lack of signal peptide cleavage in this system. Importantly, identification of P101 by direct analysis of its export function relied neither on prior identification of the mycoplasmal product nor on complete expression of the product from the cloned mycoplasma gene.
Insights
Researchers identified a Mycoplasma hyorhinis protein (P101) that can cross bacterial membranes. This finding overcomes challenges in studying mycoplasma proteins, even with problematic UGA codons, advancing our understanding of host-microbe interactions.
Area of Science:
- Microbiology
- Molecular Biology
- Protein Translocation
Background:
- Mycoplasma proteins crossing the plasma membrane are crucial for host interactions.
- Studying these proteins is difficult due to a lack of mycoplasma genetic systems and issues with translating UGA codons in other bacteria.
Purpose of the Study:
- To directly analyze a Mycoplasma hyorhinis gene encoding a protein (P101) that facilitates its own membrane translocation.
- To overcome limitations in studying mycoplasma protein function and export.
Main Methods:
- Screening a phage genomic library with antibodies from an arthritis-affected host.
- Using Tn5 and deletion mutations for protein localization.
- Employing lambda::TnphoA transposon mutagenesis in Escherichia coli to assess protein translocation.
- Cell fractionation and detergent phase partitioning to determine protein localization in E. coli.
Main Results:
- A gene for a hydrophilic protein (P101) with three UGA codons and a unique signal peptide was identified.
- E. coli expressing p101 fusions translocated up to 48 kDa of P101 across its plasma membrane.
- Fusion proteins contained mycoplasma epitopes and were integral membrane proteins in E. coli, indicating no signal peptide cleavage.
- Successful identification of P101 relied on direct export function analysis, not prior product identification or complete gene expression.
Conclusions:
- The P101 protein from Mycoplasma hyorhinis possesses intrinsic membrane translocation capabilities.
- This study demonstrates a viable method for analyzing mycoplasma protein export functions in E. coli, circumventing genetic system limitations.
- The findings facilitate the study of wall-less microbes' interactions with hosts.