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Nucleoside diphosphatase (NDPase) activity associated with human beta-protein amyloid fibers

H M Wisniewski1, A W Vorbrodt, M H Epstein

  • 1New York State Office of Mental Retardation and Developmental Disabilities, Institute for Basic Research in Developmental Disabilities, Staten Island 10314.

Acta Neuropathologica
|January 1, 1991
PubMed

Insights

Nucleoside diphosphatase (NDPase) activity in aged human brain microglial cells (MCs) suggests a role in amyloid protein elaboration. Similar mechanisms may be involved in scrapie, linking NDPase to amyloid glycosylation.

Area of Science:

  • Neuroscience
  • Cell Biology
  • Biochemistry

Background:

  • Microglial cells (MCs) are implicated in brain aging and neurodegenerative diseases.
  • Amyloid plaques are a hallmark of Alzheimer's disease and other amyloidopathies.
  • Nucleoside diphosphatase (NDPase) is an enzyme with diverse cellular functions.

Purpose of the Study:

  • To investigate the role of NDPase in the context of amyloid plaque formation in aged human cerebral cortices.
  • To explore the relationship between microglial cells, amyloid fibers, and NDPase activity.
  • To compare findings in human brain with those in scrapie-infected mouse models.

Main Methods:

  • Electron microscope cytochemistry was employed on surgical specimens of aged human cerebral cortices.
  • Localization of NDPase activity was examined at the cellular and subcellular levels.
  • Observations were compared with existing data from scrapie-infected mouse brains.

Main Results:

  • NDPase activity was detected on the surface of microglial cells (MCs).
  • Significant NDPase activity was found within endoplasmic reticulum (ER) cisternae containing amyloid fibers, continuous with extracellular deposits.
  • A close structural association between MCs and amyloid plaques was observed, suggesting MCs' involvement in amyloid elaboration.

Conclusions:

  • NDPase may play a role in the final elaboration of amyloid protein in the human brain.
  • Similar mechanisms involving NDPase and microglial cells might be conserved across species and diseases, including scrapie.
  • The co-localization of NDPase with amyloid fibers suggests a potential link to glycosyltransferase activities involved in amyloid or amyloid precursor protein glycosylation.

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