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Related Concept Videos

Spinal Cord Injury ll: Pathophysiology01:14

Spinal Cord Injury ll: Pathophysiology

Spinal cord injury progresses through two interconnected phases: primary injury and secondary injury.Primary InjuryPrimary injury happens at the moment of trauma and involves immediate mechanical damage to the spinal cord.Compression happens when broken vertebrae, herniated discs, or accumulating blood (such as a hematoma) press directly against the spinal cord, distorting its normal shape and function. In cases of contusion, the cord is bruised by a blunt force (like penetrating injuries or...
Cranial and Spinal Meninges01:19

Cranial and Spinal Meninges

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Cranial Meninges
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Hemorrhagic Stroke ll: Pathophysiology

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Increased Intracranial Pressure ll: Pathophysiology01:29

Increased Intracranial Pressure ll: Pathophysiology

Increased intracranial pressure (ICP) refers to a potentially life-threatening rise in pressure inside the skull. This usually happens when there is a major change in the volume of brain tissue, blood, or cerebrospinal fluid (CSF) — the three components inside the skull. According to the Monro-Kellie doctrine, if the volume of one component increases, the volumes of the other components must decrease to maintain normal pressure. If this does not happen, ICP rises.The process often begins with...
Hemorrhagic Stroke l: Introduction01:17

Hemorrhagic Stroke l: Introduction

A hemorrhagic stroke is an acute neurological event that occurs when a weakened cerebral blood vessel ruptures, allowing blood to accumulate within or around the brain. The sudden release of blood forms a focal hematoma that increases intracranial pressure, displaces neural tissue, and can obstruct cerebrospinal fluid pathways. These effects may be compounded by intraventricular extension of the hemorrhage, cerebral edema, or compression of adjacent structures, all of which contribute to...
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Accessing the Porcine Brain via High-Speed Pneumatic Drill Craniectomy
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Published on: July 5, 2024

Dural laceration.

Rob D Dickerman, Ashley S Reynolds, Jennifer Tackett

    Journal of Neurosurgery. Spine
    |July 2, 2008
    PubMed
    Summary
    This summary is machine-generated.

    Delayed dural laceration by hydroxyapatite (HA) spacers can cause tetraparesis after double-door laminoplasty. This complication, resulting from HA spacer dislodgement, requires surgical repair for symptom improvement.

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    Expansion Duroplasty For Severe Cervical Spinal Cord Swelling After Traumatic Injury: A Step-by-Step Surgical Protocol
    05:45

    Expansion Duroplasty For Severe Cervical Spinal Cord Swelling After Traumatic Injury: A Step-by-Step Surgical Protocol

    Published on: May 26, 2026

    Area of Science:

    • Neurosurgery
    • Spinal Surgery
    • Orthopedic Surgery

    Background:

    • Double-door laminoplasty is a surgical procedure for cervical myelopathy.
    • Complications like C-5 paralysis and axial symptoms are known.
    • Delayed dural laceration by hydroxyapatite (HA) spacers is a rare but serious complication.

    Discussion:

    • Four cases of delayed dural laceration by HA spacers causing tetraparesis after double-door laminoplasty are presented.
    • The mechanism involves anterior dislodgement of HA spacers, leading to dural laceration and clinical aggravation.
    • This complication can occur due to spacer loosening or suture breakage before bone fusion.

    Key Insights:

    • Anterior dislodgement of HA spacers can cause delayed dural laceration.
    • T2-weighted MRI is crucial for diagnosing HA spacer dislodgement and surrounding cerebrospinal fluid.
    • Surgical removal of dislodged HA spacers and dural repair leads to symptom improvement.

    Outlook:

    • Consider HA spacer dislodgement as a potential late complication of double-door laminoplasty.
    • Further research into preventing HA spacer migration is warranted.
    • Early diagnosis and surgical intervention are key to managing this complication.