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Related Concept Videos

Peptic Ulcer01:27

Peptic Ulcer

Peptic ulcers are erosive lesions of the gastric or duodenal lining, most commonly caused by Helicobacter pylori infection. This Gram-negative, helical bacterium has adapted to survive the stomach’s acidic environment by producing urease, which converts urea into ammonia and carbon dioxide. The ammonia neutralizes gastric acid in the bacterium’s immediate environment, allowing colonization of the gastric mucosa. H. pylori attaches to mucus-secreting epithelial cells, penetrates the mucus...
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Microbiota of the Stomach and Small Intestine

The human gastrointestinal (GI) tract is characterized by distinct physicochemical conditions that shape its microbial communities. Among these, the stomach presents a particularly challenging environment for microbial colonization due to its highly acidic pH, ranging from 1 to 3. This extreme acidity effectively limits microbial density. However, certain acid-tolerant microorganisms are capable of surviving in this niche. Notably, Helicobacter pylori can colonize the gastric mucosa,...
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Entamoeba histolytica, a protozoan parasite, is responsible for intestinal and extraintestinal amebiasis. Though a significant proportion of infections remain asymptomatic, approximately 50 million individuals annually are estimated to present with clinical disease, resulting in up to 100,000 deaths globally. The disease burden is disproportionately high in regions with lower socioeconomic status, such as parts of India, Africa, Mexico, and Latin America.Etiology and TransmissionThe infective...
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Bacterial gastroenteritis, characterized by diarrhea, abdominal cramps, and vomiting, is often caused by ingestion of contaminated food or water and is frequently associated with pathogenic Escherichia coli strains. These microbes exploit two principal mechanisms to inflict disease.Shiga toxin–producing E. coli, also referred to as STEC—notably O157:H7—release Shiga toxins that target ribosomes, blocking protein synthesis. The B subunit of the toxin binds the host glycolipid receptor...
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Helicobacter pylori, a resilient gram-negative bacterium, can thrive in the stomach's harsh, acidic environment. Infection with H. pylori leads to a cascade of events within the stomach lining. One of the critical disruptions caused by this bacterium is the interference with somatostatin production, a hormone responsible for regulating acid secretion. This interference tips the balance, escalating acid secretion and diminishing bicarbonate levels. This imbalance compromises the defensive...
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The gut microbiome is formed by a vast and diverse community of bacteria that colonizes our large intestine. These bacteria start residing in the gut from birth and continue diversifying throughout life, influenced by factors such as diet, lifestyle, and stress. The gut bacterial community also includes bacteria from food and those that enter the colon through the anus.
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Updated: Jul 3, 2026

Mouse Models Of Helicobacter Infection And Gastric Pathologies
07:43

Mouse Models Of Helicobacter Infection And Gastric Pathologies

Published on: October 18, 2018

Helicobacter equorum is highly prevalent in foals.

H Moyaert1, F Haesebrouck, J Dewulf

  • 1Department of Pathology, Bacteriology and Avian Diseases, Faculty of Veterinary Medicine, Ghent University, Salisburylaan 133, B-9820 Merelbeke, Belgium. Hilde.Moyaert@UGent.be

Veterinary Microbiology
|July 16, 2008
PubMed
Summary

Helicobacter equorum DNA was detected in over half of foals aged 1-6 months, indicating a higher prevalence in young horses compared to adults. Further research is needed to determine if this bacterium causes intestinal disease in foals.

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Area of Science:

  • Veterinary Microbiology
  • Equine Health
  • Bacterial Pathogenesis

Background:

  • Helicobacter equorum is an enterohepatic Helicobacter species found in horses.
  • Previous studies indicated a low prevalence of H. equorum in adult horses.

Purpose of the Study:

  • To investigate the prevalence of Helicobacter equorum in foals of different age groups.
  • To compare the prevalence of H. equorum in foals with that in adult horses.

Main Methods:

  • Faecal samples were collected from 66 foals aged 3 days to 6 months.
  • Polymerase chain reaction (PCR) was used to detect H. equorum DNA by amplifying a 1074 bp fragment of the 23S rRNA gene.

Main Results:

  • H. equorum DNA was detected in 28.6% of foals less than 1 month old.
  • A significantly higher prevalence of 67.8% was observed in foals aged 1 to 6 months.
  • This contrasts with the 0.8-7.9% prevalence found in adult horses.

Conclusions:

  • The prevalence of H. equorum in horses is age-dependent, being significantly higher in foals aged 1-6 months.
  • The high prevalence in young foals suggests a potential role in intestinal health, warranting further investigation into disease association.