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Related Concept Videos

Hemorrhagic Stroke ll: Pathophysiology01:29

Hemorrhagic Stroke ll: Pathophysiology

A hemorrhagic stroke develops when a cerebral blood vessel ruptures, allowing blood to escape into the surrounding brain tissue, as in intracerebral hemorrhage (ICH), or into the subarachnoid space, as in subarachnoid hemorrhage (SAH). Because the skull is a rigid compartment, the sudden presence of extravascular blood rapidly increases intracranial pressure and compresses adjacent neural structures, leading to immediate tissue injury and impaired cerebral perfusion.Mass Effect and Primary...
Ischemic Stroke ll: Pathophysiology01:15

Ischemic Stroke ll: Pathophysiology

An ischemic stroke occurs when a cerebral blood vessel becomes obstructed, most often by a thrombus or embolus, interrupting the delivery of oxygen and glucose to brain tissue. Because neurons rely on continuous aerobic metabolism, energy failure begins within minutes of reduced perfusion. The region receiving the least blood flow becomes the infarct core, an area of irreversible cellular death. Surrounding this core lies the penumbra, a zone of hypoperfused but still viable tissue that is...
Hemorrhagic Stroke l: Introduction01:17

Hemorrhagic Stroke l: Introduction

A hemorrhagic stroke is an acute neurological event that occurs when a weakened cerebral blood vessel ruptures, allowing blood to accumulate within or around the brain. The sudden release of blood forms a focal hematoma that increases intracranial pressure, displaces neural tissue, and can obstruct cerebrospinal fluid pathways. These effects may be compounded by intraventricular extension of the hemorrhage, cerebral edema, or compression of adjacent structures, all of which contribute to...
Ischemic Stroke l: Introduction01:15

Ischemic Stroke l: Introduction

Ischemic stroke is an acute cerebrovascular condition in which blood flow to a brain region is suddenly interrupted, leading to tissue infarction. Neurons depend on continuous oxygen and glucose supply, so even brief reductions in perfusion cause energy failure, ionic imbalance, and irreversible injury. Ischemic strokes are classified into thrombotic and embolic types based on their underlying mechanisms.Thrombotic MechanismsThrombotic stroke develops when a clot forms within a cerebral artery.
Stroke: Introduction and Types01:29

Stroke: Introduction and Types

A stroke is an acute neurological event caused by the sudden disruption of cerebral blood flow, leading to rapid loss of neuronal function. Neurons depend on continuous oxygen and glucose supply, so even brief interruptions can cause irreversible injury within minutes. Strokes are classified into ischemic and hemorrhagic types.Ischemic StrokeIschemic strokes are most common and occur due to arterial occlusion, depriving brain tissue of oxygen and nutrients. This leads to energy failure, ionic...
Alterations in Muscle Tone ll01:12

Alterations in Muscle Tone ll

Alterations in muscle tone are common manifestations of neurological disorders and reflect dysfunction within different nervous system regions. Spasticity, paratonia, and dystonia represent distinct forms of hypertonia, each with unique mechanisms, clinical features, and diagnostic importance.CharacteristicsSpasticity happens from upper motor neuron lesions and is characterized by velocity-dependent resistance to passive movement. Clinical features include:Exaggerated deep tendon reflexesClonus...

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Related Experiment Video

Updated: Jul 2, 2026

Kinematic Analysis Using 3D Motion Capture of Drinking Task in People With and Without Upper-extremity Impairments
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Published on: March 28, 2018

[Movement disorders in stroke].

J Ghika1

  • 1Service de neurologie, CHUV, Lausanne, Suisse. joseph.ghika@rsv-gnw.ch

Revue Neurologique
|September 2, 2008
PubMed
Summary
This summary is machine-generated.

Dyskinesias, or involuntary movements, are uncommon in acute stroke but can occur later. Specific types like limb shaking and hemichorea-hemiballism suggest stroke-related motor pathway damage.

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Dual-Task Stroop Paradigm for Detecting Cognitive Deficits in High-Functioning Stroke Patients
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Last Updated: Jul 2, 2026

Kinematic Analysis Using 3D Motion Capture of Drinking Task in People With and Without Upper-extremity Impairments
08:45

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Published on: March 28, 2018

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Area of Science:

  • Neurology
  • Neuroscience
  • Movement Disorders

Context:

  • Dyskinesias are infrequent in acute stroke (1%) but may present later.
  • The full spectrum of hyper- and hypo-akinetic syndromes is known, yet specific stroke-related presentations are less defined.
  • Literature lacks prevalence data for delayed dyskinesia post-stroke.

Purpose:

  • To explore the specific manifestations and potential underlying mechanisms of dyskinesias in acute and delayed stroke presentations.
  • To identify key neurological pathways and brain structures implicated in stroke-induced dyskinesias.
  • To understand the role of brain plasticity in the late development of dyskinesias after stroke.

Summary:

  • Three dyskinetic syndromes—limb shaking, hemichorea-hemiballism, and unilateral asterixis—are particularly indicative of acute stroke.
  • Lesions associated with these dyskinesias span the frontosubcortical motor circuit, including cortex, basal ganglia, thalamus, cerebellum, and brainstem.
  • Limb shaking may relate to transient frontosubcortical pathway ischemia, while other dyskinesias involve diverse stroke locations (ischemic/hemorrhagic).

Impact:

  • Highlights specific motor syndromes as potential early indicators of stroke.
  • Elucidates the broad range of brain structures affected by stroke-induced movement disorders.
  • Suggests that late-onset dyskinesias may represent adaptive motor control strategies developed through neuroplasticity post-stroke.