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Exposure to ultrafine particles in asphalt work
Karine Elihn1, Bente Ulvestad, Siri Hetland
1Department of Applied Environmental Science, Stockholm University, Stockholm, Sweden. Karine.Elihn@itm.su.se
Journal of Occupational and Environmental Hygiene
|October 14, 2008
Summary
Asphalt workers experience lung function loss due to inflammation, primarily from ultrafine particles generated during paving. These particles, originating from asphalt fumes and traffic, pose potential health risks.
Area of Science:
- Occupational Health
- Environmental Science
- Pulmonary Medicine
Background:
- Epidemiologic studies show asphalt workers have reduced lung function and increased inflammation markers.
- The specific agents causing inflammation in asphalt paving workers require further investigation.
Purpose of the Study:
- To identify agents responsible for inflammatory reactions in asphalt workers.
- To specifically assess the role of ultrafine particles in these inflammatory responses.
Main Methods:
- Personal sampling for total dust, polycyclic aromatic hydrocarbons, and nitrogen dioxide (NO2).
- Measurement of ultrafine particle exposure using particle counters and a scanning mobility particle sizer.
- Analysis of organic and elemental carbon fractions in particles.
Main Results:
- Asphalt pavers were exposed to high concentrations of ultrafine particles (mean 3.4 x 10^4/cm^3).
- Ultrafine particles originated mainly from asphalt fumes and traffic exhaust.
- Oil mist occasionally exceeded occupational limits; NO2 and total dust remained within limits.
Conclusions:
- Asphalt paving workers face significant exposure to ultrafine particles throughout their workday.
- Ultrafine particles from asphalt fumes are a likely cause of inflammation and lung function decline.
- Occupational exposure limits for some agents were exceeded, indicating potential adverse health effects.
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