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Related Concept Videos

Hemorrhagic Stroke ll: Pathophysiology01:29

Hemorrhagic Stroke ll: Pathophysiology

A hemorrhagic stroke develops when a cerebral blood vessel ruptures, allowing blood to escape into the surrounding brain tissue, as in intracerebral hemorrhage (ICH), or into the subarachnoid space, as in subarachnoid hemorrhage (SAH). Because the skull is a rigid compartment, the sudden presence of extravascular blood rapidly increases intracranial pressure and compresses adjacent neural structures, leading to immediate tissue injury and impaired cerebral perfusion.Mass Effect and Primary...
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A brain abscess is a focal, intracerebral infection characterized by a localized collection of pus within the brain parenchyma, resulting from microbial invasion and the body’s inflammatory response. It progresses through stages: early and late cerebritis, followed by early and late capsule formation, reflecting tissue destruction, immune response, and eventual encapsulation.Etiology and PathogenesisCausative organisms vary with source and host factors, often involving polymicrobial infections,...
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Increased intracranial pressure (ICP) refers to a potentially life-threatening rise in pressure inside the skull. This usually happens when there is a major change in the volume of brain tissue, blood, or cerebrospinal fluid (CSF) — the three components inside the skull. According to the Monro-Kellie doctrine, if the volume of one component increases, the volumes of the other components must decrease to maintain normal pressure. If this does not happen, ICP rises.The process often begins with...
Increased Intracranial Pressure l: Introduction01:14

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Intracranial hypertension is a sustained elevation of intracranial pressure (ICP) above 22 mm Hg. In supine adults, normal ICP is ~7–15 mm Hg.The rigid, nonexpandable cranium contains three components—brain tissue, blood, and cerebrospinal fluid (CSF)—that total ~1,700 mL in a typical adult: 1,400 mL brain (~80%), 150 mL blood (~10%), and 150 mL CSF (~10%). According to the Monro–Kellie doctrine, total intracranial volume is effectively fixed. When one component expands, CSF and venous blood...

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Updated: Jun 27, 2026

Endoscopic Endonasal Trans-sphenoidal Approach: Minimally Invasive Surgery for Pituitary Adenomas
07:43

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Published on: January 17, 2018

Acute subdural hematoma after transsphenoidal surgery.

Girish Menon1, Biji Bahuleyan, Suresh Nair

  • 1Department of Neurosurgery, Sree Chitra Tirunal Institute for Medical Sciences and Technology, Trivandrum 695011, India. gmr@sctimst.ac.in

Journal of Clinical Neuroscience : Official Journal of the Neurosurgical Society of Australasia
|November 18, 2008
PubMed
Summary

A rare complication, acute subdural hematoma, occurred in a patient after pituitary adenoma surgery and cerebrospinal fluid management. Prompt recognition and intervention are crucial for managing this serious neurosurgical risk.

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Area of Science:

  • Neurosurgery
  • Neurology
  • Endocrinology

Background:

  • Transsphenoidal decompression is a common surgical approach for pituitary adenomas.
  • Postoperative cerebrospinal fluid (CSF) management, including lumbar drains, is sometimes employed to address CSF leaks.
  • Acute subdural hematoma (ASH) is a rare but serious complication following neurosurgical procedures.

Observation:

  • A 48-year-old male developed an acute subdural hematoma after transsphenoidal decompression for a pituitary adenoma.
  • The patient had a lumbar drain placed to manage postoperative cerebrospinal fluid rhinorrhea.
  • The subdural hematoma presented acutely following the surgical intervention and CSF management.

Findings:

  • The case highlights a potential, albeit uncommon, risk associated with transsphenoidal surgery and subsequent lumbar drain placement.
  • Discussion of risk factors contributing to the development of acute subdural hematoma in this context is presented.
  • The study emphasizes the critical importance of vigilant patient monitoring and timely diagnostic imaging.

Implications:

  • Early detection and prompt surgical or medical intervention are vital for improving outcomes in patients developing acute subdural hematoma postoperatively.
  • This case underscores the need for careful consideration of potential complications, even with established surgical techniques.
  • Neurosurgeons and neurologists should maintain a high index of suspicion for subdural hematoma in patients with neurological decline after pituitary surgery and CSF diversion.