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Microbicidal activities of Salmonella typhimurium- and interferon-gamma-activated mouse peritoneal macrophages

J A Langermans1, P H Nibbering, M E van der Hulst

  • 1Department of Infectious Diseases, University Hospital, Leiden, The Netherlands.

Insights

Infection activates mouse macrophages, but this does not guarantee enhanced bacterial killing. Recombinant interferon-gamma (rIFN-gamma) alone is insufficient to boost macrophage listericidal activity.

Area of Science:

  • Immunology
  • Microbiology
  • Cell Biology

Background:

  • Peritoneal macrophages play a crucial role in the innate immune response against intracellular pathogens.
  • Macrophage activation is a complex process that can be triggered by various stimuli, including microbial infections and cytokines like interferon-gamma.
  • Assessing macrophage activation requires multiple functional criteria, such as pathogen growth inhibition, reactive oxygen species production, and antigen expression.

Purpose of the Study:

  • To investigate the activation status of mouse peritoneal macrophages during infections with facultative intracellular bacteria.
  • To evaluate the impact of recombinant interferon-gamma (rIFN-gamma) on macrophage activation and bactericidal activity.
  • To determine if activated macrophages exhibit enhanced killing of specific bacterial pathogens like Salmonella and Listeria.

Main Methods:

  • Infection of mice with Salmonella typhimurium and Listeria monocytogenes.
  • Intravenous injection of recombinant interferon-gamma (rIFN-gamma) into mice.
  • Assessment of macrophage activation using criteria: inhibition of Toxoplasma gondii proliferation, enhanced hydrogen peroxide (H2O2) release, and increased Ia antigen expression.
  • Quantification of bacterial proliferation rates in spleen and liver tissues.
  • Evaluation of bactericidal activity against Salmonella and Listeria.

Main Results:

  • Macrophages activated during Salmonella typhimurium infection did not show enhanced killing of Salmonella or Listeria.
  • Listeria-activated macrophages exhibited increased killing of Listeria but not Salmonella.
  • Recombinant interferon-gamma (rIFN-gamma) administration did not enhance macrophage killing of Salmonella or Listeria.
  • Bacterial proliferation rates in spleen and liver were comparable between activated and control mice for Salmonella.
  • Listeria proliferation was reduced in the liver of rIFN-gamma-treated mice only when low bacterial doses (≤1 LD50) were administered.

Conclusions:

  • Peritoneal macrophages become activated during infections with intracellular pathogens.
  • Macrophage activation does not confer enhanced bactericidal activity against all bacterial species.
  • Recombinant interferon-gamma (rIFN-gamma) alone is not sufficient to enhance the listericidal activity of macrophages in vivo.

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