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Posterior cricoarytenoid muscle denervation
S Kano1, J B Horowitz, C T Sasaki
1Section of Otolaryngology, Yale University School of Medicine, New Haven, Conn. 06510.
Archives of Otolaryngology--Head & Neck Surgery
|September 1, 1991
Summary
Vocal fold paralysis often stems from recurrent laryngeal nerve injury. This study found that canine posterior cricoarytenoid muscles do not show atrophy for at least one year after denervation, aiding reinnervation procedure planning.
Area of Science:
- Veterinary Medicine
- Laryngology
- Neurology
Background:
- Vocal fold paralysis commonly results from recurrent laryngeal nerve injury.
- Understanding denervation atrophy timelines is crucial for planning reinnervation surgeries.
- Limited long-term data exists on canine posterior cricoarytenoid muscle changes post-denervation.
Purpose of the Study:
- To investigate the timeline of muscle atrophy in the canine posterior cricoarytenoid muscle after denervation.
- To provide data for optimizing reinnervation strategies in cases of vocal fold paralysis.
Main Methods:
- Surgical denervation of the recurrent laryngeal nerve in canine subjects.
- Histological and functional assessments of the posterior cricoarytenoid muscle over a one-year period.
Main Results:
- No significant muscle atrophy was observed in the denervated posterior cricoarytenoid muscles at the 1-year mark.
- Muscle structure and function remained relatively preserved in the absence of nerve input for up to one year.
Conclusions:
- Canine posterior cricoarytenoid muscle exhibits resilience to denervation atrophy for at least one year.
- This extended period without atrophy offers a significant window for successful reinnervation procedures following vocal fold paralysis.