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Scanning electron microscopic observations on muscle cells of experimental mitochondrial myopathy produced by 2,

H Kawahara1, S Houdou, T Inoué

  • 1Division of Child Neurology, Tottori University School of Medicine, Yonago, Japan.

Journal of Submicroscopic Cytology and Pathology
|July 1, 1991
PubMed

Insights

Scanning electron microscopy revealed significant mitochondrial and sarcoplasmic reticulum changes in experimental myopathy. This study highlights novel sarcoplasmic reticulum alterations in mitochondrial myopathy.

Area of Science:

  • Cell Biology
  • Muscle Physiology
  • Pathology

Background:

  • Skeletal muscle cells undergo morphological changes in myopathies.
  • 2,4-dinitrophenol can induce experimental myopathy.
  • Scanning electron microscopy (SEM) offers 3D visualization of cellular ultrastructure.

Purpose of the Study:

  • To examine the morphological changes in skeletal muscle cells of rats with experimental myopathy induced by 2,4-dinitrophenol.
  • To compare these changes with the ultrastructure of normal muscle cells using SEM.
  • To elucidate the three-dimensional structure of intracellular components in myopathic muscle.

Main Methods:

  • Induction of experimental myopathy in rats using 2,4-dinitrophenol.
  • Preparation of muscle specimens using the Aldehyde-Osmium-DMSO-Osmium method for SEM.
  • Three-dimensional imaging of intracellular structures, including mitochondria and sarcoplasmic reticulum.

Main Results:

  • Large accumulations of mitochondria were observed in the subsarcolemmal region of myopathic muscle cells.
  • Mitochondria showed swelling and abnormal concentric cristae in perinuclear and intermyofibrillar regions.
  • The sarcoplasmic reticulum network, present in normal cells, was absent in the experimental mitochondrial myopathy.

Conclusions:

  • The study provides a detailed 3D ultrastructural analysis of experimental mitochondrial myopathy using SEM.
  • Observed mitochondrial changes are consistent with previous transmission electron microscopy findings.
  • Significant and previously undescribed alterations in the sarcoplasmic reticulum were identified in this myopathy model.

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