Androgenic regulation of hedgehog signaling pathway components in prostate cancer cells

Mengqian Chen1, Matthew Tanner, Alice C Levine

  • 1Cancer Center, The Ordway Research Institute, 150 New Scotland Avenue, Albany, NY 12208, USA.

Insights

Androgen deprivation therapy for prostate cancer may unexpectedly activate hedgehog signaling. This pathway activation, driven by low androgen levels, could impact treatment effectiveness, suggesting combined therapies.

Area of Science:

  • Oncology
  • Molecular Biology
  • Cell Signaling

Background:

  • Hedgehog signaling is implicated in various human cancers, including prostate cancer.
  • Prostate cancer cells express hedgehog pathway components but are typically unresponsive to canonical hedgehog signaling.
  • Androgen's role in regulating hedgehog signaling in prostate cancer remains unclear.

Purpose of the Study:

  • To investigate the regulation of hedgehog signaling by androgen in prostate cancer cells.
  • To determine if androgen deprivation influences hedgehog ligand expression and pathway activity.
  • To explore the potential implications for prostate cancer treatment.

Main Methods:

  • Utilized LNCaP and its metastatic variants (C4-2, C4-2B) human prostate cancer cell lines.
  • Assessed hedgehog ligand and target gene expression under varying androgen conditions (R1881 treatment, androgen deprivation).
  • Measured hedgehog ligand release into conditioned medium and its effect on responsive cells; analyzed Gli target gene expression and cyclopamine sensitivity.

Main Results:

  • Androgen (R1881) suppressed hedgehog ligand expression in prostate cancer cells.
  • Prolonged androgen deprivation significantly upregulated Sonic and Indian hedgehog mRNA and protein (up to 30,000-fold).
  • Androgen-deprived LNCaP cell medium activated hedgehog signaling in mouse fibroblasts and increased Gli target gene expression (Patched 1, Gli2) in LNCaP cells, which was reversible by cyclopamine.

Conclusions:

  • Androgen modulates hedgehog ligand expression and autocrine hedgehog signaling in prostate cancer cells.
  • Chronic androgen deprivation may create a pro-hedgehog signaling environment, potentially affecting treatment efficacy.
  • Clinical trials combining hedgehog-blocking drugs with androgen deprivation therapy for advanced prostate cancer are warranted.

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