Related Experiment Video
Updated: Jun 25, 2026

08:20
Simultaneous Application of Transcranial Direct Current Stimulation during Virtual Reality Exposure
Published on: January 18, 2021
How long do the short-term violent video game effects last?
Christopher Barlett1, Omar Branch, Christopher Rodeheffer
1Department of psychology, Iowa State University, Ames, Iowa 50011, USA. cpb6666@iastate.edur 2009
Aggressive Behavior
|February 12, 2009
Summary
Violent video games temporarily increase aggression and arousal. These effects, including aggressive feelings and thoughts, fade within 4 minutes, while physical responses and aggressive behaviors persist for 4-9 minutes.
Area of Science:
- Psychology
- Media Psychology
- Cognitive Psychology
Background:
- Violent video games are popular and their effects on aggression are widely studied.
- Previous research indicates short-term increases in aggression and physiological arousal after gameplay.
- The duration of these effects requires further investigation.
Purpose of the Study:
- To determine the lingering effects of violent video game play on aggression and physiological arousal.
- To investigate the time course of aggressive feelings, thoughts, behaviors, and heart rate after gameplay.
Main Methods:
- Two studies were conducted with participants (N=91 each) playing violent video games.
- Measures included aggressive thoughts, feelings, heart rate, and aggressive behavior (hot sauce paradigm).
- Participants were assessed at varying delay intervals (0-10 minutes) post-gameplay.
Main Results:
- Violent video game play initially increased aggressive feelings, thoughts, behavior, and heart rate.
- Aggressive feelings and thoughts returned to baseline within 4 minutes.
- Heart rate and aggressive behavior remained elevated for 4-9 minutes.
Conclusions:
- The immediate effects of violent video games on aggression and arousal are short-lived.
- While cognitive and emotional aggression markers dissipate quickly, physiological arousal and behavioral aggression persist longer.
- Findings suggest differential recovery rates for various aggression-related outcomes.
Related Concept Videos
Traumatic Brain Injury l: Introduction
DefinitionTraumatic brain injury, or TBI, is a disturbance of normal brain function induced by an external mechanical force, such as a direct blow to the head or a penetrating injury. It can affect both brain structure and function, producing a wide range of clinical outcomes. TBI is a heterogeneous condition, meaning its effects may differ based on the type, location, and severity of the injury.Basis of ClassificationTBI is classified based on severity, injury mechanism, or pathophysiology. In...
Traumatic Memory
Emotionally traumatic events often lead to memories that are exceptionally vivid and enduring, sometimes persisting with remarkable clarity throughout an individual's life. A classic example of this phenomenon is a person who survives a car accident. Even years later, they may recall every detail of the event with startling accuracy — the screeching of the tires, the jarring impact, and the acrid smell of burning rubber. Such vividness contrasts sharply with how an individual remembers mundane...
Vascular Spasm
The vascular phase, also known as vasospasm, is the initial stage of hemostasis, crucial for preventing excessive bleeding when a blood vessel is injured. After a vessel is cut, nerves in the damaged area trigger pain and other sensory impulses. Simultaneously, the smooth muscles in the vessel wall contract, resulting in a vascular spasm. This contraction reduces the vessel's diameter at the injury site, slowing or stopping blood loss through the vessel wall. Vascular spasms typically last for...
Secondary Spinal Cord Injury llI: Pathophysiology
Early Ischemia and Ionic ImbalanceWithin minutes of spinal cord injury, a secondary cascade begins, progressing over hours to weeks. Vascular damage reduces blood flow, causing ischemia and mitochondrial dysfunction. ATP depletion leads to ion pump failure, membrane depolarization, sodium influx, potassium efflux, and water accumulation, resulting in cellular swelling. Increased intracellular calcium further disrupts mitochondria and accelerates cellular injury.Excitotoxicity and Neuronal...
