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Blood laboratory findings in patients suffering from self-perceived electromagnetic hypersensitivity (EHS)
Norbert Dahmen1, David Ghezel-Ahmadi, Alice Engel
1Department of Psychiatry, University of Mainz, Germany.
Bioelectromagnetics
|March 5, 2009
Summary
Electromagnetic hypersensitivity (EHS) may be linked to common somatic issues. This study found thyroid, liver, and inflammatory markers in some EHS individuals, suggesting treatable conditions may cause symptoms.
Area of Science:
- Environmental Health
- Clinical Medicine
- Medical Diagnostics
Background:
- Electromagnetic hypersensitivity (EHS) is characterized by unspecific symptoms attributed to electromagnetic field exposure.
- The underlying pathophysiology of EHS remains unknown, posing therapeutic challenges.
- Common somatic health problems may contribute to symptoms experienced by electrosensitive individuals.
Purpose of the Study:
- To investigate potential common somatic health problems in individuals with EHS.
- To analyze clinical laboratory parameters in EHS sufferers and compare them to controls.
- To identify if specific laboratory abnormalities correlate with EHS symptoms.
Main Methods:
- Analysis of clinical laboratory parameters: thyroid-stimulating hormone (TSH), alanine transaminase (ALT), aspartate transaminase (AST), creatinine, hemoglobin, hematocrit, and C-reactive protein (CRP).
- Comparison of laboratory results between 132 EHS patients (42 males, 90 females) and 101 controls (34 males, 67 females).
- Statistical analysis to identify significant differences in laboratory markers between the EHS group and the control group.
Main Results:
- Laboratory signs of thyroid dysfunction, liver dysfunction, and chronic inflammatory processes were identified in notable fractions of EHS sufferers.
- Significant differences in TSH and ALT/AST levels were observed between EHS patients and controls.
- The study refuted hypotheses linking anemia or kidney dysfunction to EHS.
Conclusions:
- Common somatic conditions, such as thyroid and liver dysfunction or chronic inflammation, may underlie symptoms in some individuals with EHS.
- Further investigation into these identified laboratory abnormalities is warranted.
- Clinical recommendation: screen individuals with self-proclaimed EHS for treatable somatic conditions.
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