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Light and electron microscopic studies of pathologic changes induced in mice by ciguatoxin poisoning

K Terao1, E Ito, M Oarada

  • 1Research Center for Pathogenic Fungi and Microbial Toxicoses, Chiba University, Japan.

Insights

Ciguatoxin and ciguatoxin-4c cause acute poisoning in mice, affecting the heart, adrenal glands, and nerves. These toxins lead to cardiac cell damage and adrenal medulla degeneration, with some mice experiencing penile erection.

Area of Science:

  • Toxicology
  • Pathology
  • Marine Biotoxins

Background:

  • Ciguatoxins are potent marine phycotoxins responsible for ciguatera fish poisoning.
  • Understanding the toxicological mechanisms and target organs of ciguatoxins is crucial for clinical management.

Purpose of the Study:

  • To investigate the acute toxic effects and target organs of ciguatoxin and ciguatoxin-4c in a mouse model.
  • To examine the pathological alterations induced by these toxins using light and electron microscopy.

Main Methods:

  • Male ICR mice were administered ciguatoxin or ciguatoxin-4c via intraperitoneal injection or oral gavage.
  • Histopathological examination using light and electron microscopy was performed on various organs, including the heart, adrenal glands, autonomic nerves, and penis.

Main Results:

  • No significant difference in toxicity was observed between ciguatoxin and ciguatoxin-4c.
  • Cardiac muscle cells showed swelling and focal necrosis, with interstitial effusion.
  • Degeneration was noted in the adrenal medulla, and continuous penile erection occurred in approximately 15% of affected mice.
  • Autonomic nerve fibers and synapses were affected, but intestinal mucosa and muscle layers remained largely unaltered.

Conclusions:

  • Ciguatoxin and ciguatoxin-4c induce acute toxic effects primarily targeting the cardiovascular system, adrenal medulla, and autonomic nervous system.
  • Atropine partially mitigated diarrhea, while reserpine exacerbated symptoms, suggesting complex neuroautonomic involvement.
  • Further research is needed to elucidate the precise mechanisms of ciguatoxin-induced pathology.

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