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Spinal epidural hematoma in hemophilic children: controversies in management
Pilar Varela Rois1, Manuel Rodríguez López, Beatriz Cabanas López de Vergara
1Department of Neurosurgery, Xeral-Cies Hospital, Pizarro Street, Vigo 36204, Pontevedra, Spain. pilirois@hotmail.com
Insights
Spinal epidural hematoma (SEH) in children with hemophilia requires prompt treatment. Aggressive factor replacement and surgical decompression offer a safe and effective approach for managing SEH and preventing neurological deficits.
Area of Science:
- Pediatric Hematology
- Neurosurgery
- Spinal Surgery
Background:
- Spinal epidural hematoma (SEH) is a rare but serious complication in children with hemophilia.
- SEH can lead to rapid and severe neurological deficits.
Observation:
- A 13-year-old boy with hemophilia A presented with acute spinal pain and weakness without reported trauma.
- MRI confirmed an extradural mass from D5 to D6.
- The patient underwent emergency hemilaminectomies and clot evacuation.
Findings:
- Aggressive recombinant factor VIII replacement therapy was initiated.
- Surgical decompression resulted in an excellent recovery.
- Early diagnosis and intervention are crucial for favorable outcomes.
Implications:
- This case highlights the importance of timely diagnosis and multidisciplinary management for SEH in hemophilic children.
- Prompt surgical decompression is a safe and effective treatment for selected cases.
- Aggressive factor replacement therapy is essential in managing SEH in this patient population.
Introduction And Objective:
Spinal epidural hematoma (SEH) is an uncommon complication in hemophilic children. It can produce rapidly progressive neurological deficits. We aim to discuss the different management options for these patients.
Case Report:
A 13-year-old boy with a history of hemophilia A was admitted with acute onset of localized spine pain and weakness. No trauma was reported on review of the history. Recombinant factor VIII aggressive replacement therapy was started. Spinal magnetic resonance imaging revealed an extradural mass lesion extending from D5 to D6 level. Emergency hemilaminectomies of D5 and D6 and evacuation of the clot were done. The patient made excellent recovery following surgery.
Conclusion:
Early diagnosis and immediate aggressive replacement therapy are mandatory in the management of SEH. Prompt surgical decompression to avoid any permanent neurological deficit is a safe and effective treatment option for an SEH in selected hemophilic children.
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