Endothelial dysfunction--an obstacle of therapeutic angiogenesis

Lan Sun1, Yongyi Bai, Guanhua Du

  • 1National Center for Pharmaceutical Screening, Institute of Materia Medica, Chinese Academy of Medical Science and Peking Union Medical College, Beijing, China. muyeliuyunyi@gmail.com

Insights

Therapeutic angiogenesis aims to grow new blood vessels for ischemic cardiovascular disease. However, clinical trials fail due to endothelial dysfunction in older patients, unlike young animals used in studies.

Area of Science:

  • Cardiovascular Medicine
  • Regenerative Medicine
  • Molecular Biology

Background:

  • Increasing numbers of patients with ischemic cardiovascular disease are not candidates for revascularization.
  • Therapeutic angiogenesis, using growth factors to promote collateral blood vessel development, is a key research area.
  • Pre-clinical successes in animal models have not translated to clinical trial efficacy.

Purpose of the Study:

  • To review therapeutic angiogenesis trials.
  • To discuss mechanisms of impaired angiogenic response in endothelial dysfunction.
  • To explore endothelial function-improving strategies to enhance angiogenesis.

Main Methods:

  • Retrospective analysis of therapeutic angiogenesis clinical trials.
  • Review of pre-clinical studies in animal models.
  • Examination of mechanisms underlying endothelial dysfunction and angiogenic impairment.

Main Results:

  • Clinical trials of therapeutic angiogenesis have yielded limited success.
  • Animal studies often use healthy young subjects, unlike older patients with risk factors in trials.
  • Endothelial dysfunction in older patients may significantly reduce the effectiveness of growth factor therapy.

Conclusions:

  • Endothelial dysfunction is a primary reason for the failure of therapeutic angiogenesis trials.
  • Strategies to improve endothelial function may enhance angiogenic responses.
  • Further research is needed to optimize angiogenic therapies for diverse patient populations.

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