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Open Angle Glaucoma: Treatment

Open Angle Glaucoma: Treatment

In open-angle glaucoma, the iridocorneal angle remains open, but the trabecular meshwork becomes stiff, slowing down the outflow of aqueous humor. This causes a buildup of aqueous humor in the anterior chamber, leading to a sudden increase in intraocular pressure. The treatment for open-angle glaucoma focuses on reducing the elevated intraocular pressure by either decreasing the secretion of aqueous humor or increasing its outflow.
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Glaucoma: Overview

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Glaucoma is an eye condition characterized by increased intraocular pressure that damages the retina and optic nerve, leading to irreversible blindness if left untreated. The human eye has various components, including the cornea, iris, pupil, lens, and optic nerve. Aqueous humor is secreted by the epithelium of the ciliary body in the posterior chamber and flows through the trabecular meshwork and canal of Schlemm, maintaining normal intraocular pressure. The trabecular meshwork and the canal...

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Angle Closure Glaucoma: Treatment

Angle-closure glaucoma, or closed-angle glaucoma, is an eye condition where the iris bulges out and blocks the iridocorneal angle, resulting in a buildup of aqueous humor and increased intraocular pressure. Immediate medical attention is necessary due to the sudden onset of symptoms. The treatment for angle-closure glaucoma includes short-term and long-term approaches. Short-term treatment involves using eye drops like pilocarpine to lower intraocular pressure by increasing aqueous humor...

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Related Experiment Video

Updated: Jun 21, 2026

Assessing Early Stage Open-Angle Glaucoma in Patients by Isolated-Check Visual Evoked Potential

Assessing Early Stage Open-Angle Glaucoma in Patients by Isolated-Check Visual Evoked Potential

Published on: May 25, 2020

[Glaucoma diagnosis using scanning laser polarimetry].

E M Hoffmann¹, A Schulze

¹Univ.-Augenklinik Mainz, Universitätsmedizin Mainz, Deutschland. ehoffman@uni-mainz.de

Der Ophthalmologe : Zeitschrift Der Deutschen Ophthalmologischen Gesellschaft

|August 5, 2009

Summary

Scanning laser polarimetry (SLP) aids early glaucoma detection by measuring retinal nerve fiber layer (RNFL) thickness. Enhanced corneal compensation (ECC) technology improves diagnostic accuracy by addressing atypical birefringence patterns.

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Last Updated: Jun 21, 2026

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Multimodal Volumetric Retinal Imaging by Oblique Scanning Laser Ophthalmoscopy (oSLO) and Optical Coherence Tomography (OCT)

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Doppler Optical Coherence Tomography of Retinal Circulation

Doppler Optical Coherence Tomography of Retinal Circulation

Published on: September 18, 2012

Area of Science:

Ophthalmology and Medical Optics
Biomedical Engineering

Context:

Early glaucoma diagnosis is crucial for preventing vision loss.
Scanning laser polarimetry (SLP) is an established imaging technique for assessing the retinal nerve fiber layer (RNFL).
Previous SLP devices faced limitations due to atypical birefringence patterns causing inaccurate RNFL thickness measurements.

Purpose:

To evaluate the efficacy of enhanced corneal compensation (ECC) technology in Scanning Laser Polarimetry (SLP) for improving glaucoma diagnosis.
To assess the impact of ECC on differentiating between healthy eyes and those with glaucoma, particularly in the presence of atypical birefringence.

Summary:

Scanning laser polarimetry (SLP) measures retinal nerve fiber layer (RNFL) thickness using polarimetry.
Variable corneal compensation and the newer enhanced corneal compensation (ECC) technology aim to improve the differentiation between glaucoma and healthy eyes.
ECC technology specifically addresses atypical birefringence patterns that can lead to false high RNFL values, enhancing diagnostic accuracy.

Impact:

Improved accuracy in early glaucoma diagnosis through advanced RNFL thickness measurement.
Potential for more reliable patient follow-up and trend analysis with ECC-enabled SLP.
Further long-term studies are needed to fully establish the long-term effectiveness and clinical utility of ECC in glaucoma management.