[Rupture of ileal varices in a type C liver cirrhosis patient: a case report]

Takahisa Suzuki1, Mutsumi Murayama, Masataka Shinoda

  • 1Department of Gastroenterogy, Toyota Memorial Hospital, Japan. takahisa@mail.toyota.co.jp

Insights

Bleeding from small intestinal varices is rare but should be considered in liver cirrhosis patients with unexplained melena. This case highlights ileal varices as a potential source of massive lower gastrointestinal bleeding.

Area of Science:

  • Gastroenterology
  • Vascular Surgery

Background:

  • Liver cirrhosis frequently leads to gastrointestinal bleeding, typically from esophageal varices.
  • Non-variceal sources of bleeding in cirrhosis patients can be challenging to diagnose.

Observation:

  • A 74-year-old woman with liver cirrhosis presented with recurrent massive lower gastrointestinal bleeding.
  • Imaging revealed ileal varices (small intestinal varices) and associated collateral veins.

Findings:

  • Ileal varices were surgically resected, with exposed vessels on the mucosal surface identified as the bleeding source.
  • This case demonstrates small intestinal varices as a cause of significant gastrointestinal hemorrhage.

Implications:

  • Small intestinal varices should be included in the differential diagnosis for obscure gastrointestinal bleeding in patients with liver cirrhosis.
  • Advanced imaging and surgical intervention may be necessary for diagnosis and management.

Related Concept Videos

Esophageal Varices-I: Introduction01:24

Esophageal Varices-I: Introduction

Esophageal varices are dilated, tortuous veins which are found mainly in the submucosa of the lower esophagus but which may also appear higher up or extend into the stomach. They develop due to increased pressure in the portal venous system, often as a result of liver cirrhosis. This condition scars and damages the liver, impeding normal blood flow through the portal vein. To compensate, blood seeks alternative pathways, forming fragile new vessels (varices) in the esophagus and stomach. These...
Esophageal Varices-II: Clinical Features and Management01:28

Esophageal Varices-II: Clinical Features and Management

Esophageal varices often manifest as gastrointestinal bleeding episodes, presenting symptoms like hematemesis (vomiting of blood), hematochezia (passing fresh blood via the rectum), and melena (black, tarry stools). Other signs can include weight loss, anorexia, abdominal discomfort, jaundice, pruritus, altered mental status, and muscle cramps.
In the initial assessment, a thorough review of the patient's medical history is vital to identify risk factors such as liver disease, alcohol abuse, or...
Portal Hypertension01:22

Portal Hypertension

Portal hypertension is an increase in blood pressure within the portal venous system. Normally, this pressure is less than 5 mmHg. It is considered clinically significant when it rises above 10 mmHg. At this threshold, complications from altered blood flow and venous congestion emerge.EtiologyPortal hypertension arises from conditions that impede blood flow through the liver. The most common cause is cirrhosis, in which chronic liver injury leads to fibrotic scarring. This fibrosis narrows or...
Cirrhosis I: Introduction01:23

Cirrhosis I: Introduction

Cirrhosis is a chronic, irreversible liver disease characterized by the widespread replacement of healthy liver tissue with fibrotic scar tissue and the formation of regenerative nodules.Etiology of cirrhosisCirrhosis results from sustained liver injury that triggers progressive fibrosis and structural remodeling. The underlying causes are diverse, encompassing common and less frequent clinical conditions. Regardless of the origin, all causes lead to chronic inflammation, hepatocyte loss, and...
Cirrhosis II: Pathophysiology01:24

Cirrhosis II: Pathophysiology

Cirrhosis is a progressive chronic liver injury caused by prolonged inflammation, excessive fibrotic remodeling, and impaired regeneration. Over time, repeated hepatic insults disrupt the liver’s architecture and function, leading to reduced blood flow, impaired bile drainage, and diminished metabolic capacity.Pathophysiology of cirrhosisCirrhosis arises from three main responses to chronic liver damage: inflammation, immune activation, and hepatocyte death. These processes lead to structural...