Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Pulmonary Hypertension: Classification and Pathogenesis01:30

Pulmonary Hypertension: Classification and Pathogenesis

Pulmonary hypertension (PH) is a severe health condition in which the mean pulmonary arterial pressure increases to 25 mmHg or more, even when the body is at rest. This high pressure in the blood vessels that transport blood from the heart to the lungs can cause various symptoms, including shortness of breath, can lead to right heart failure, and significantly affect the overall quality of life.
There are various classifications for PH, each relating to different underlying causes and also...
Atelectasis II: Pathophysiology01:10

Atelectasis II: Pathophysiology

Atelectasis develops when alveoli lose their air and collapse inward. Because lung tissue is naturally elastic, these air sacs shrink rather than remaining open. Collapsed alveoli are no longer ventilated, reducing their role in gas exchange. Blood flow may continue in these regions, creating a ventilation–perfusion mismatch. Clinical findings include decreased breath sounds, dullness to percussion, reduced chest expansion, and decreased tactile fremitus as sound transmission through collapsed...
Acute Respiratory Failure-II01:21

Acute Respiratory Failure-II

Type I Respiratory Failure, or hypoxemic respiratory failure, occurs when the partial pressure of oxygen (PaO2) in arterial blood falls below 60 mmHg while breathing room air without a corresponding increase in arterial carbon dioxide levels (PaCO2). This condition highlights a significant impairment in the lungs' capacity to oxygenate the blood.
The underlying physiological abnormalities that contribute to hypoxemic respiratory failure include:
Chronic Obstructive Pulmonary Disease II: Emphysema01:23

Chronic Obstructive Pulmonary Disease II: Emphysema

Emphysema, a major phenotype of chronic obstructive pulmonary disease (COPD), is characterized by irreversible destruction of alveolar walls and permanent enlargement of distal airspaces. Unlike chronic bronchitis, which primarily affects the airways, emphysema predominantly involves the lung parenchyma, where structural damage leads to airflow limitation.PathophysiologyIt most commonly results from prolonged exposure to cigarette smoke and other toxic gases, particularly cigarette smoke.
Chronic Obstructive Pulmonary Disease III: Chronic Bronchitis Features01:24

Chronic Obstructive Pulmonary Disease III: Chronic Bronchitis Features

Chronic bronchitis is a key phenotype of chronic obstructive pulmonary disease (COPD), characterized by airway-centered inflammation and mucus overproduction. It develops from long-term exposure to harmful particles or gases, most commonly cigarette smoke, which triggers a persistent inflammatory response.Cellular and Structural ChangesInflammation initially affects the large bronchi and later the smaller airways, with infiltration by immune cells, including neutrophils, macrophages, and...
Pulmonary Embolism I: Introduction01:29

Pulmonary Embolism I: Introduction

Pulmonary embolism (PE) occurs when a thrombus, fat or air embolus, amniotic fluid, or tumor tissue blocks one or more pulmonary arteries. These blockages originate in the venous system or the right side of the heart.EtiologyPE primarily arises from deep vein thrombosis (DVT) and other hypercoagulable states, such as inherited thrombophilias. Additional etiological factors include venous stasis, commonly seen in obesity, and endothelial injury from surgery and trauma. Less common causes include...

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

How should we manage fatigue in on-call workers? A review of guidance materials and a systematic review of the evidence-base.

Sleep medicine reviews·2024
Same author

Optical Manipulation of Bipolarons in a System with Nonlinear Electron-Phonon Coupling.

Physical review letters·2024
Same author

Significance of immunohematologic testing in mother and newborn ABO incompatibility.

Immunohematology·2023
Same author

Updated S2 K guidelines for the management of bullous pemphigoid initiated by the European Academy of Dermatology and Venereology (EADV).

Journal of the European Academy of Dermatology and Venereology : JEADV·2022
Same author

Platelet transfusion practice and related transfusion reactions in a large teaching hospital.

Transfusion clinique et biologique : journal de la Societe francaise de transfusion sanguine·2021
Same author

Acute hemolytic transfusion reaction caused by anti-Yt<sup>a</sup>.

Immunohematology·2021
Same journal

[Satisfaction of young doctors in Croatia: are we heading in the right direction?}.

Lijecnicki vjesnik·2018
Same journal

[Empa-reg and leader trials -revolution in modern diabetology?].

Lijecnicki vjesnik·2018
Same journal

[AUTONOMIC DYSREFLEXIA].

Lijecnicki vjesnik·2018
Same journal

[ASSESSMENT OF QUALITY OF LIFE IN GLAUCOMA PATIENTS].

Lijecnicki vjesnik·2018
Same journal

[HBsAg SEROCONVERSION IN THE TREATMENT OF CHRONIC HEPATITIS B WITH PEGYLATED INTERFERON].

Lijecnicki vjesnik·2018
Same journal

[FEMUR FRACTURES IN CHILDREN – CAUSES AND MECHANISMS OF INJURY].

Lijecnicki vjesnik·2018
See all related articles

Related Experiment Video

Updated: Jun 21, 2026

Increasing Pulmonary Artery Pulsatile Flow Improves Hypoxic Pulmonary Hypertension in Piglets
08:08

Increasing Pulmonary Artery Pulsatile Flow Improves Hypoxic Pulmonary Hypertension in Piglets

Published on: May 11, 2015

[Pulmonary hypoplasia].

M Raos1, I Koncul, K Kovac

  • 1Specijalna bolnica za bolesti disnog sustava djece i mladezi, Zagreb.

Lijecnicki Vjesnik
|August 8, 2009
PubMed
Summary
This summary is machine-generated.

Pulmonary hypoplasia, a rare lung development disorder, was diagnosed in a child with persistent respiratory issues after pneumonia. The condition stemmed from a hypoplastic left pulmonary artery, impacting left lung development.

More Related Videos

Chronic Thromboembolic Pulmonary Hypertension and Assessment of Right Ventricular Function in the Piglet
09:22

Chronic Thromboembolic Pulmonary Hypertension and Assessment of Right Ventricular Function in the Piglet

Published on: November 4, 2015

Related Experiment Videos

Last Updated: Jun 21, 2026

Increasing Pulmonary Artery Pulsatile Flow Improves Hypoxic Pulmonary Hypertension in Piglets
08:08

Increasing Pulmonary Artery Pulsatile Flow Improves Hypoxic Pulmonary Hypertension in Piglets

Published on: May 11, 2015

Chronic Thromboembolic Pulmonary Hypertension and Assessment of Right Ventricular Function in the Piglet
09:22

Chronic Thromboembolic Pulmonary Hypertension and Assessment of Right Ventricular Function in the Piglet

Published on: November 4, 2015

Area of Science:

  • Pediatric Pulmonology
  • Medical Imaging
  • Congenital Lung Abnormalities

Background:

  • Pneumonia can sometimes mask or coexist with underlying congenital lung abnormalities.
  • Persistent auscultatory signs after infection resolution warrant further investigation for developmental issues.

Observation:

  • A pediatric patient presented with persistent auscultatory findings post-pneumonia treatment.
  • Radiological imaging revealed left lung hyperlucency and diminished vascularity.
  • Perfusion scintigraphy demonstrated absent perfusion in the affected left lung.

Findings:

  • Bronchography indicated reduced left lung volume and bronchiectasis.
  • Cerebral angiography identified a hypoplastic left pulmonary artery as the primary cause.
  • The constellation of findings confirmed pulmonary hypoplasia.

Implications:

  • Early diagnosis of pulmonary hypoplasia is crucial for appropriate management.
  • Advanced imaging techniques are vital for identifying rare congenital pulmonary anomalies.
  • Understanding the vascular origin of lung dysgenesis aids in clinical diagnosis.