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Related Concept Videos

Cardiomyopathy III: Hypertrophic Cardiomyopathy01:29

Cardiomyopathy III: Hypertrophic Cardiomyopathy

Hypertrophic cardiomyopathy, or HCM, is an autosomal dominant genetic disorder characterized by asymmetric left ventricular hypertrophy without ventricular dilation. It is more common in men and is typically diagnosed in young, athletic adults.EtiologyHCM is primarily genetic and is caused by mutations in genes encoding sarcomeric proteins. Researchers have identified over 1400 mutations across at least 11 different genes. Among these, the most frequently occurring mutations are found in the...
Mitral Stenosis I: Introduction01:22

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Mitral Valve Stenosis (MVS) is a heart condition where the mitral valve narrows, impeding blood circulation from the left atrium to the left ventricle. The etiology and pathophysiology of this condition are multifaceted, leading to a cascade of cardiovascular complications.Causes of Mitral Valve StenosisRheumatic Heart Disease: It is the main cause of mitral valve stenosis, particularly in developing nations. This condition arises from rheumatic fever, an inflammatory illness resulting from...
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Cardiomyopathy IV: Restrictive Cardiomyopathy

Restrictive cardiomyopathy (RCM) is a rare heart muscle disease characterized by impaired ventricular filling due to stiffened ventricular walls, leading to significant diastolic dysfunction.EtiologyRestrictive cardiomyopathy can arise from both inherited and acquired diseases, many of which are systemic. It is categorized into four main types: infiltrative, storage, non-infiltrative, and endomyocardial diseases.Infiltrative diseases, such as amyloidosis, lead to RCM by depositing amyloid...
Mitral Valve Prolapse I: Introduction01:27

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Dilated cardiomyopathy, or DCM, is a progressive myocardial disorder characterized by ventricular chamber dilation and contractile dysfunction.EtiologyVarious factors can cause DCM, including hypertension and heavy alcohol intake, which contribute to the weakening and enlargement of the heart muscle. Viral infections, such as Coxsackievirus B, adenoviruses, and influenza, can lead to DCM by causing inflammation and damage to heart tissue. Certain chemotherapeutic agents, including daunorubicin,...
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Related Experiment Video

Updated: Jun 20, 2026

Murine Fetal Echocardiography
08:04

Murine Fetal Echocardiography

Published on: February 15, 2013

Left ventricular noncompaction associated with Ebstein's anomaly.

Levent Saltik1, Elif Eroğlu, Fatih Bayrak

  • 1Department of Pediatric Cardiology, Cerrahpaşa Medicine Faculty of Istanbul University, Istanbul, Turkey.

Turk Kardiyoloji Dernegi Arsivi : Turk Kardiyoloji Derneginin Yayin Organidir
|September 1, 2009
PubMed
Summary

Left ventricular noncompaction, a rare congenital heart defect, was diagnosed in a young boy with Ebstein's anomaly. Regular follow-up confirmed his asymptomatic status and preserved cardiac function.

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Morphological and Functional Assessment of the Right Ventricle Using 3D Echocardiography
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Last Updated: Jun 20, 2026

Murine Fetal Echocardiography
08:04

Murine Fetal Echocardiography

Published on: February 15, 2013

Morphological and Functional Assessment of the Right Ventricle Using 3D Echocardiography
07:11

Morphological and Functional Assessment of the Right Ventricle Using 3D Echocardiography

Published on: October 28, 2020

Area of Science:

  • Cardiology
  • Pediatric Cardiology
  • Congenital Heart Disease

Background:

  • Left ventricular noncompaction (LVNC) is a rare congenital cardiomyopathy.
  • It is characterized by prominent myocardial trabeculations and deep recesses.
  • Association with Ebstein's anomaly is exceptionally rare.

Observation:

  • A 13-year-old boy presented with a cardiac murmur.
  • Echocardiography revealed LVNC with normal LV size and ejection fraction.
  • Mild tricuspid regurgitation and right atrial enlargement were noted, consistent with Ebstein's anomaly.

Findings:

  • The patient remained asymptomatic with preserved biventricular systolic function over a five-year follow-up.
  • No significant progression of LVNC or associated cardiac abnormalities was observed.
  • Low-dose aspirin was the only prescribed medication.

Implications:

  • This case highlights the rare co-occurrence of LVNC and Ebstein's anomaly.
  • It underscores the importance of echocardiographic evaluation in asymptomatic individuals with congenital heart defects.
  • Long-term monitoring is crucial for managing patients with noncompaction cardiomyopathy, even in the absence of symptoms.