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Related Concept Videos

Narcolepsy01:07

Narcolepsy

Narcolepsy is a chronic sleep disorder characterized by pervasive, uncontrolled sleepiness and other sleep disturbances. One of its hallmark symptoms is an abrupt transition to REM sleep upon falling asleep, which causes symptoms typically associated with this phase to occur unexpectedly during wakefulness. These include the following symptoms, which typically last from a minute or two to half an hour.
Botulism01:22

Botulism

Botulism is a life-threatening neuroparalytic condition caused by botulinum neurotoxin, which is produced by the bacterium Clostridium botulinum, a Gram-positive, spore-forming, obligate anaerobe.In adults, the toxin enters the body in different ways: in foodborne botulism, the preformed toxin is absorbed in the intestine. In wound botulism, spores grow in injured tissue and release the toxin into the blood. Infant botulism differs mechanistically from adult forms. In infants, botulism commonly...

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Related Experiment Video

Updated: Jun 20, 2026

Multi-system Monitoring for Identification of Seizures, Arrhythmias and Apnea in Conscious Restrained Rabbits
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Published on: March 27, 2021

[Organic catatonia after apoplexy].

Anders Jørgensen1, Martin Balslev Jørgensen

  • 1Psykiatrisk Center, Rigshospitalet, DK-2100 København Ø. aj@dadlnet.dk

Ugeskrift for Laeger
|September 8, 2009
PubMed
Summary

Catatonia can manifest in psychiatric disorders and rarely in organic brain conditions. This case highlights catatonia following a right-hemisphere stroke, responding to treatment.

Area of Science:

  • Neurology
  • Psychiatry

Background:

  • Catatonia is a neuropsychiatric syndrome associated with various conditions.
  • Organic cerebral affections can rarely present with catatonia.

Observation:

  • A previously healthy male developed catatonia years after a right-hemisphere stroke.
  • Symptoms included stupor, mutism, stereotyped movements, and echolalia.

Findings:

  • The patient's catatonic symptoms responded to benzodiazepine and electroconvulsive therapy.
  • This suggests a potential link between focal cerebral lesions and catatonia.

Implications:

  • Catatonia should be considered in the differential diagnosis of patients with known organic brain disorders.
  • Further research is needed to understand the neurobiological mechanisms underlying stroke-induced catatonia.

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