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Demonstration of bacterial antigen in macrophages in experimental pyelonephritis

B Iványi1, T Krenács, E Dobó

  • 1Department of Pathology, Albert Szent-Györgyi Medical University, Szeged, Hungary.

Virchows Archiv. B, Cell Pathology Including Molecular Pathology
|January 1, 1990
PubMed

Insights

Interstitial macrophages in rats with Escherichia coli pyelonephritis store bacterial residues for weeks. This indicates impaired phagolysosomal function in these PAS-positive macrophages, hindering bacterial clearance.

Area of Science:

  • Immunology
  • Pathology
  • Microbiology

Background:

  • Experimental Escherichia coli pyelonephritis in rats is a model for studying bacterial kidney infections.
  • Interstitial macrophages play a crucial role in the host immune response to bacterial pathogens.

Purpose of the Study:

  • To investigate the immunomorphological characteristics of interstitial macrophages containing Periodic Acid-Schiff (PAS)-positive granules during experimental Escherichia coli pyelonephritis.
  • To determine the fate of bacterial components within these macrophages over time.

Main Methods:

  • Utilized immunohistochemistry, immunoelectron microscopy, light microscopy, and electron microscopy.
  • Examined tissue samples at twelve time points, from 2 days to 13 weeks post-infection.
  • Employed an anti-E. coli antibody to trace bacterial antigens.

Main Results:

  • Macrophages with PAS-positive granules appeared by day 7 and persisted.
  • These granules contained phagolysosomes filled with myelin figures derived from bacterial cell walls.
  • Bacterial residues with intact antigenic structures were retained for up to 13 weeks, reacting with the anti-E. coli antibody.
  • This suggests a failure in phagolysosomal elimination of bacterial substances.

Conclusions:

  • PAS-positive macrophages in pyelonephritis exhibit disturbed phagolysosomal elimination of bacterial residues.
  • Lysosomal overloading with bacteria and debris likely leads to enzyme depletion and incomplete bacterial breakdown.
  • This retention of bacterial antigens may prolong the inflammatory process or indicate persistent bacterial presence.

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