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Related Concept Videos

Urinary Tract Calculi V: Nursing Management01:28

Urinary Tract Calculi V: Nursing Management

AssessmentSubjective Data: Obtain a detailed health history, including any recent or chronic urinary tract infections, periods of immobilization, previous episodes of renal calculi, and medical conditions such as gout, benign prostatic hyperplasia, or hyperparathyroidism. Review the medication history for drugs that may influence stone formation, including allopurinol, analgesics, loop diuretics, or thiazide diuretics. Document the use of long-term indwelling catheters and any past surgical...
Urinary Tract Calculi III: Medical Management01:30

Urinary Tract Calculi III: Medical Management

The diagnosis of renal calculi involves several imaging techniques, including non-contrast CT scans and ultrasound. These methods help visualize kidney stones, assess their size and location, and detect possible obstructions. Additionally, Measuring urine pH is useful for diagnosing specific stone types, such as struvite (alkaline pH) and uric acid stones (acidic pH). Cystine stones are primarily linked to cystinuria, a genetic condition. A urinalysis helps detect blood in the urine (hematuria)...
Acute Pyelonephritis II: Diagnostic Studies and Management01:28

Acute Pyelonephritis II: Diagnostic Studies and Management

Introduction:For diagnosing acute pyelonephritis, a comprehensive patient history is collected to identify symptoms such as dysuria, frequent or urgent urination, flank pain, or costovertebral angle (CVA) tenderness that may suggest a kidney infection.Physical ExaminationDuring the physical examination, CVA tenderness is assessed. This involves gentle percussion over the costovertebral angle, where tenderness often indicates a kidney infection.Diagnostic TestsUrinalysis: Used to identify white...
Nephrotic Syndrome II : Assessment and Medical Management01:26

Nephrotic Syndrome II : Assessment and Medical Management

IntroductionNephrotic syndrome is a kidney disorder marked by excessive protein loss in the urine, leading to various systemic complications. This condition often results from damage to the glomeruli—the kidney's filtering units—causing proteinuria, low blood protein levels, and fluid retention. Understanding the assessment, diagnosis, and management of nephrotic syndrome is essential for effective treatment and prevention of further kidney damage.AssessmentPatient History: Document any history...
Gastritis III: Clinical Manifestations and Management01:23

Gastritis III: Clinical Manifestations and Management

The clinical manifestations of gastritis can vary depending on the cause and type of gastritis, but some common symptoms may include the following.
Clinical manifestations of acute gastritis
The patient with acute gastritis may have a rapid onset of symptoms, such as epigastric pain or discomfort, dyspepsia, anorexia, hiccups, or nausea and vomiting, which can last from a few hours to a few days. Erosive or hemorrhagic gastritis may cause bleeding, which may manifest as blood in vomit or as...
Acute Pancreatitis II: Clinical Manifestations and Management01:30

Acute Pancreatitis II: Clinical Manifestations and Management

Acute pancreatitis presents a complex medical emergency characterized by rapid onset inflammation of the pancreas, demanding timely diagnosis and management to prevent complications. The condition primarily manifests through severe upper abdominal pain that often radiates to the back. This pain intensifies following the consumption of fatty foods. Accompanying symptoms such as nausea, vomiting, abdominal distention, fever, dyspnea, cyanosis, and jaundice can vary in intensity but significantly...

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Related Experiment Videos

Gout--current diagnosis and treatment.

Anne-Kathrin Tausche1, Tim L Jansen, Hans-Egbert Schröder

  • 1Bereich Rheumatologie, Medizinische Klinik und Poliklinik III, Universitätsklinikum, Carl Gustav Carus an der TU Dresden, Dresden, Germany. anne-kathrin.tausche@uniklinikum-dresden.de

Deutsches Arzteblatt International
|October 2, 2009
PubMed
Summary

Gout treatment requires managing acute attacks with NSAIDs, colchicine, or steroids, followed by long-term uric acid lowering to prevent crystal buildup and structural damage. The goal is serum uric acid below 360 micromol/L.

Keywords:
allopurinolgouthyperuricemia,deposits of uric acidtreatment

Related Experiment Videos

Area of Science:

  • Rheumatology
  • Metabolic Disorders
  • Pharmacology

Background:

  • Hyperuricemia is common in aging populations, often co-occurring with metabolic disorders.
  • Elevated serum uric acid can lead to gout, characterized by crystal deposition and inflammation.
  • Gout prevalence in Germany is 1.4%, with no specific national treatment guidelines.

Purpose of the Study:

  • To review the diagnosis and treatment of gout.
  • To provide evidence-based recommendations for gout management.

Main Methods:

  • A selective literature review was conducted.
  • The review focused on diagnostic criteria and therapeutic interventions for gout.

Main Results:

  • Asymptomatic hyperuricemia typically does not require drug treatment.
  • Acute gouty arthritis attacks are effectively treated with NSAIDs, colchicine, or glucocorticosteroids.
  • Long-term management aims to reduce uric acid crystal deposits and maintain serum levels below 360 micromol/L.

Conclusions:

  • Clinical gout necessitates addressing hyperuricemia to prevent recurrent inflammation and structural damage.
  • Long-term urate-lowering therapy, using agents like allopurinol or uricosurics, is crucial.
  • Effective gout management involves both acute symptom control and chronic uric acid reduction.