Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Transient Ischemic Attack l: Introduction01:26

Transient Ischemic Attack l: Introduction

A transient ischemic attack (TIA) is a brief episode of neurological dysfunction caused by a temporary, focal reduction in cerebral blood flow. Although symptoms resemble those of an ischemic stroke, the interruption in perfusion is short-lived and does not cause permanent infarction. TIAs are clinically important because they often serve as early warning events for future stroke.Mechanisms of Transient Cerebral IschemiaTransient cerebral ischemia may arise through several mechanisms. One...
Ischemic Stroke l: Introduction01:15

Ischemic Stroke l: Introduction

Ischemic stroke is an acute cerebrovascular condition in which blood flow to a brain region is suddenly interrupted, leading to tissue infarction. Neurons depend on continuous oxygen and glucose supply, so even brief reductions in perfusion cause energy failure, ionic imbalance, and irreversible injury. Ischemic strokes are classified into thrombotic and embolic types based on their underlying mechanisms.Thrombotic MechanismsThrombotic stroke develops when a clot forms within a cerebral artery.
Ischemic Stroke ll: Pathophysiology01:15

Ischemic Stroke ll: Pathophysiology

An ischemic stroke occurs when a cerebral blood vessel becomes obstructed, most often by a thrombus or embolus, interrupting the delivery of oxygen and glucose to brain tissue. Because neurons rely on continuous aerobic metabolism, energy failure begins within minutes of reduced perfusion. The region receiving the least blood flow becomes the infarct core, an area of irreversible cellular death. Surrounding this core lies the penumbra, a zone of hypoperfused but still viable tissue that is...

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Ultra-deep <i>vs</i>. standard swept-source optical coherence tomography for posterior vitreous detachment assessment: a retrospective comparative study.

Quantitative imaging in medicine and surgery·2026
Same author

Bilingual evaluation of large language models for patient education in refractive surgery.

International journal of ophthalmology·2026
Same author

Modulation of Respiration-Triggered Trans-Spinal Magnetic Stimulation on Diaphragmatic Motor-Evoked Potentials Following Cervical Spinal Cord Contusion in Rats.

Journal of neurotrauma·2026
Same author

Risk Factors for Diabetic Retinopathy Change With Diabetes Duration: Synergistic Effect of Long Duration and Anemia.

Journal of diabetes research·2026
Same author

Taiwan Clinical Practice Guidelines for Myasthenia Gravis.

Acta neurologica Taiwanica·2026
Same author

Natural History and Progression of Untreated Keratoconus: An Updated Systematic Review and Meta-Analysis.

Current eye research·2026

Related Experiment Video

Updated: Jun 19, 2026

Modeling Stroke in Mice: Permanent Coagulation of the Distal Middle Cerebral Artery
08:14

Modeling Stroke in Mice: Permanent Coagulation of the Distal Middle Cerebral Artery

Published on: July 31, 2014

Isolated ataxia after pure left insular cortex infarction.

Li-Min Liou1, Yuh-Cherng Guo, Chiou-Lian Lai

  • 1Department of Neurology, Kaohsiung Municipal Hsiao-kang Hospital, Kaohsiung Medical University, Kaohsiung, Taiwan.

Neurological Sciences : Official Journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology
|October 7, 2009
PubMed
Summary

Acute insular cortex infarction can cause various symptoms. This case highlights a link between left insular infarction and truncal ataxia, suggesting a new diagnostic consideration.

More Related Videos

Performing Permanent Distal Middle Cerebral with Common Carotid Artery Occlusion in Aged Rats to Study Cortical Ischemia with Sustained Disability
09:11

Performing Permanent Distal Middle Cerebral with Common Carotid Artery Occlusion in Aged Rats to Study Cortical Ischemia with Sustained Disability

Published on: February 23, 2016

Related Experiment Videos

Last Updated: Jun 19, 2026

Modeling Stroke in Mice: Permanent Coagulation of the Distal Middle Cerebral Artery
08:14

Modeling Stroke in Mice: Permanent Coagulation of the Distal Middle Cerebral Artery

Published on: July 31, 2014

Performing Permanent Distal Middle Cerebral with Common Carotid Artery Occlusion in Aged Rats to Study Cortical Ischemia with Sustained Disability
09:11

Performing Permanent Distal Middle Cerebral with Common Carotid Artery Occlusion in Aged Rats to Study Cortical Ischemia with Sustained Disability

Published on: February 23, 2016

Area of Science:

  • Neurology
  • Neuroanatomy

Background:

  • The insular cortex is anatomically complex with diverse connections.
  • Insular infarction can lead to varied neurological deficits.

Observation:

  • A case of pure left insular cortex infarction presented with isolated truncal ataxia.
  • This symptom was not previously reported in literature for insular lesions.

Findings:

  • The study demonstrates a significant relationship between the left insular cortex and the cerebellar system.
  • Potential pathways include the spinocerebellar and dentatorubrothalamic tracts.

Implications:

  • Left insular cortex lesions should be considered in diagnosing isolated truncal ataxia.
  • This finding expands the clinical spectrum of insular infarction and its cerebellar connections.