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Plasma lecithin-cholesterol acyltransferase activity and plasma lipoprotein composition and concentrations in

M A Dhansay1, A J Benadé, P R Donald

  • 1Research Institute for Nutritional Diseases, South African Medical Research Council, Tygerberg.

Insights

Children with kwashiorkor have reduced lecithin-cholesterol acyltransferase (LCAT) activity, impacting lipoprotein composition and cholesterol ester levels. Treatment improves LCAT activity and normalizes lipoprotein profiles.

Area of Science:

  • Biochemistry
  • Pediatric Nutrition
  • Lipid Metabolism

Background:

  • Kwashiorkor, a severe form of malnutrition, is associated with significant metabolic disturbances.
  • Lipoprotein metabolism plays a crucial role in lipid transport and cardiovascular health.

Purpose of the Study:

  • To investigate plasma lecithin-cholesterol acyltransferase (LCAT) activity, lipoprotein composition, and concentrations in children with kwashiorkor.
  • To assess the impact of kwashiorkor and its treatment on these parameters.

Main Methods:

  • Measurements of plasma LCAT activity, total cholesterol, cholesterol ester, and lipoprotein composition (VLDL, IDL, LDL, HDL).
  • Analysis of apolipoprotein A-I (apo A-I) and apolipoprotein B (apo B) concentrations.
  • Longitudinal study design with measurements at baseline (day 1), during treatment (day 10), and post-treatment (day 30).

Main Results:

  • Significantly decreased LCAT activity was observed on day 1 of kwashiorkor compared to days 10 and 30.
  • Plasma total cholesterol, cholesterol esters, and lipoprotein cholesterol esters were reduced in kwashiorkor.
  • Abnormal HDL composition and reduced LDL particle number, depleted of cholesterol esters, were noted in kwashiorkor.

Conclusions:

  • Reduced plasma LCAT activity is a key factor contributing to abnormal lipoprotein composition and concentrations in children with kwashiorkor.
  • Treatment of kwashiorkor leads to improvements in LCAT activity and normalization of lipoprotein profiles.
  • These findings highlight the critical role of LCAT in managing lipid abnormalities associated with severe malnutrition.

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