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Related Experiment Video

Updated: Jun 18, 2026

Compensatory Limb Use and Behavioral Assessment of Motor Skill Learning Following Sensorimotor Cortex Injury in a Mouse Model of Ischemic Stroke
08:01

Compensatory Limb Use and Behavioral Assessment of Motor Skill Learning Following Sensorimotor Cortex Injury in a Mouse Model of Ischemic Stroke

Published on: July 10, 2014

Phantom limb after stroke: an underreported phenomenon.

Daniel Antoniello1, Benzi M Kluger, Daniel H Sahlein

  • 1Department of Neurology, Albert Einstein College of Medicine, NY, United States. dantonie@montefiore.org

Cortex; a Journal Devoted to the Study of the Nervous System and Behavior
|November 17, 2009
PubMed
Summary
This summary is machine-generated.

Phantom limb experiences are common after stroke, affecting over half of individuals studied. These sensations, including illusions of limb position and movement, suggest stroke impacts sensory and motor pathways.

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Area of Science:

  • Neuroscience
  • Neurology
  • Rehabilitation Medicine

Background:

  • Phantom limb (PL) after cerebral lesions is considered rare.
  • Previous studies have not systematically evaluated PL prevalence post-stroke.

Purpose of the Study:

  • To investigate the prevalence and characteristics of phantom limb experiences in post-stroke individuals.
  • To characterize sensory and motor features of phantom limbs following cerebral lesions.

Main Methods:

  • A structured interview and questionnaire were administered to 50 post-stroke individuals.
  • Systematic examination of perceptual characteristics of phantom limbs was conducted.

Main Results:

  • Phantom experiences were reported by over half of participants (n=27).
  • Postural phantoms (limb position illusions) and kinesthetic phantoms (illusory movements) were common.
  • A unique syndrome of volitional control over phantom movements was observed in four participants with plegic hands.

Conclusions:

  • Stroke commonly results in phantom limb experiences, contrary to previous assumptions.
  • Subtotal deafferentation or defective motor efference post-stroke may underlie these phantom sensations.