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Relation of cardiac output at rest and during exercise to age in essential hypertension

R Fagard1, J Staessen

  • 1Department of Pathophysiology, Faculty of Medicine, University of Leuven, Belgium.

Insights

Cardiac output declines with age in men with essential hypertension, irrespective of cardiovascular disease. This age-related decrease in cardiac function is independent of weight and blood pressure, suggesting other factors are at play.

Area of Science:

  • Cardiovascular Physiology
  • Gerontology
  • Hypertension Research

Background:

  • Cardiac output (CO) is known to decrease with age.
  • This decline has been attributed to an increased prevalence of occult cardiovascular disease.
  • Essential hypertension is a common condition affecting cardiovascular health.

Purpose of the Study:

  • To investigate the relationship between cardiac output and age in men with essential hypertension.
  • To determine if occult cardiovascular disease explains the age-related decline in cardiac output.
  • To analyze hemodynamic parameters across different age groups and activity levels.

Main Methods:

  • Analysis of cardiac output using the direct oxygen Fick method in 110 men aged 16-64.
  • Participants had World Health Organization stage I or II essential hypertension and were followed for 7 years.
  • Hemodynamic measurements were taken at rest (supine and seated) and during upright bicycle exercise.

Main Results:

  • Cardiac output showed a significant inverse relationship with age (p < 0.01) at rest and during exercise.
  • This inverse relationship was independent of body weight and mean intra-arterial pressure.
  • Stroke volume decreased with age in the supine position, while heart rate decreased with age during upright rest and exercise.

Conclusions:

  • In essential hypertensive men free of cardiovascular complications, cardiac output declines with age independently of activity level.
  • Occult cardiovascular disease does not appear to be the primary driver of age-related cardiac output decline in this cohort.
  • Findings suggest intrinsic aging processes or other non-disease-related factors influence cardiac output reduction over time.

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