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Pathophysiology of Citrobacter diversus neonatal meningitis: comparative studies in an infant mouse model

A L Soriano1, R G Russell, D Johnson

  • 1Department of Pediatrics, University of Maryland School of Medicine, Baltimore 21201.

Insights

Citrobacter diversus causes neonatal meningitis and brain abscesses. An infant mouse model showed age-related susceptibility and linked mortality to bacteremia, but strain-specific virulence factors remain unknown.

Area of Science:

  • * Microbiology
  • * Pathophysiology
  • * Neonatal Infections

Background:

  • * Citrobacter diversus is a significant cause of neonatal meningitis.
  • * Infections are characterized by the formation of multiple brain abscesses.
  • * Understanding the pathophysiology of these infections is crucial for developing effective treatments.

Purpose of the Study:

  • * To develop and utilize an infant mouse intracranial inoculation model.
  • * To evaluate the pathophysiology of Citrobacter diversus neonatal infections.
  • * To identify factors correlating with mortality and disease severity.

Main Methods:

  • * Intracranial inoculation of 2-day-old mice with Citrobacter diversus strains.
  • * Assessment of mortality rates and correlation with strain characteristics and bacteremia.
  • * Histopathological examination of brain lesions and evaluation of age-related susceptibility.

Main Results:

  • * High mortality (>50%) observed in 2-day-old mice inoculated with 18 of 26 C. diversus strains.
  • * Mortality significantly correlated with bacteremia, irrespective of the isolate's origin (CNS or non-CNS).
  • * Characteristic brain lesions included suppurative ventriculitis and periventricular abscessation; younger mice (5-8 days old) showed reduced susceptibility.

Conclusions:

  • * Citrobacter diversus demonstrates a propensity for causing brain abscesses in neonatal mice.
  • * Bacteremia is a key factor associated with mortality in this model.
  • * Age-related resistance to infection and CNS damage was observed, highlighting developmental factors in susceptibility.

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