Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Criteria for Causality: Bradford Hill Criteria - I01:30

Criteria for Causality: Bradford Hill Criteria - I

The Bradford Hill criteria are a group of principles that provide a framework to determine a causal relationship between a specific factor and a disease. There are nine criteria that are pivotal in assessing causality in epidemiological studies. Here's a closer look at Strength, Consistency, Specificity, and Temporality criteria with definitions and examples:
Criteria for Causality: Bradford Hill Criteria - II01:28

Criteria for Causality: Bradford Hill Criteria - II

The Bradford Hill criteria serve as guidelines for establishing causative links in epidemiological research. Beyond Strength, Consistency, Specificity, and Temporality, key criteria also include Biological Gradient, Plausibility, Coherence, Experiment, and Analogy. These principles assist scientists in assessing the likelihood of causation in complex biological contexts. Below is a summary of these concepts:
Dissociative Identity Disorder01:30

Dissociative Identity Disorder

Dissociative Identity Disorder (DID), previously termed multiple personality disorder, is a complex psychological condition characterized by the presence of two or more distinct identities or personality states. Each identity exhibits unique patterns of behavior, voice, and mannerisms and may possess separate memories and emotional responses. The alternating control between identities can result in memory gaps and challenges in recalling daily activities, often exacerbating the individual's...
Instinct Theory01:29

Instinct Theory

Instinct theory proposes that innate biological instincts, like animal behavioral patterns, primarily drive human behavior. These instincts are inborn, not learned, and are fundamental to decision-making and action. Just as animals rely on instincts for critical survival functions such as migration, nest building, and defense, humans are also believed to exhibit behaviors rooted in evolutionary needs. For example, the instinct to reproduce motivates sexual behavior, while territorial instincts...
Causes of Similarity-Dissimilarity Effect01:26

Causes of Similarity-Dissimilarity Effect

The similarity-dissimilarity effect, a fundamental concept in social psychology, explains how interpersonal similarities and differences influence attraction and social interactions. This effect is supported by three key psychological perspectives: balance theory, social comparison theory, and consensual validation.Balance Theory and Cognitive ConsistencyBalance theory, developed by Fritz Heider, posits that individuals seek cognitive consistency in their relationships. When two people share...
Theories of Dissolution: Diffusion Layer Model01:15

Theories of Dissolution: Diffusion Layer Model

Dissolution, the process by which drug particles dissolve in a solvent, is explained by the diffusion layer model, a theoretical framework that simulates the absorption of oral drugs and allows us to analyze experimental data.
This process starts with a thin layer, saturated with the drug, forming at the interface between the solid and liquid. The solute then diffuses from this layer into the main solution. The Noyes-Whitney equation suggests that the rate of dissolution relies on the diffusion...

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Medical Misadventures as Errors and Mistakes and Motor Vehicular Accidents in the Disproportionate Burden of Childhood Mortality among Blacks/African Americans in the United States: CDC Dataset, 1968-2015.

Healthcare (Basel, Switzerland)·2024
Same author

Risk Differences in Disease-Specific Infant Mortality Between Black and White US Children, 1968-2015: an Epidemiologic Investigation.

Journal of racial and ethnic health disparities·2018
Same author

LETTERS TO THE EDITOR.

Journal of the Air & Waste Management Association (1995)·2017
Same author

An Acute Respiratory Infection of a Physiologically Anemic Infant is a More Likely Cause of SIDS than Neurological Prematurity.

Frontiers in neurology·2016
Same author

RE: Overall Postneonatal Mortality and Rates of SIDS.

Pediatrics·2016
Same author

Comment on Fard et al.'s Candidate gene variants of the immune system and sudden infant death syndrome.

International journal of legal medicine·2016

Related Experiment Video

Updated: Jun 17, 2026

Functional Near-Infrared Spectroscopy Hyperscanning Study in Psychological Counseling
06:04

Functional Near-Infrared Spectroscopy Hyperscanning Study in Psychological Counseling

Published on: January 17, 2025

A Unifying Theory for SIDS.

David T Mage1, Maria Donner

  • 1A. I. duPont Hospital for Children, Biomolecular Core Laboratory, 1600 Rockland Road, Wilmington, DE 19803, USA.

International Journal of Pediatrics
|January 6, 2010
PubMed
Summary

Sudden Infant Death Syndrome (SIDS) presents distinct patterns in affected infants. A new quadruple-risk model explains these SIDS characteristics, suggesting a potential genetic link requiring further investigation.

Area of Science:

  • Pediatrics
  • Genetics
  • Epidemiology

Background:

  • Sudden Infant Death Syndrome (SIDS) remains a significant concern in infant mortality.
  • Existing theories struggle to explain the multifaceted characteristics of SIDS.
  • Understanding SIDS etiology is crucial for prevention and intervention strategies.

Purpose of the Study:

  • To present a unifying quadruple-risk model for SIDS.
  • To explain four key epidemiological characteristics of SIDS.
  • To propose a hypothesis for further confirmatory testing.

Main Methods:

  • Analysis of established SIDS epidemiological data.
  • Development of a quadruple-risk model integrating multiple factors.
  • Formulation of a genetic hypothesis for validation.

More Related Videos

Observational Study Protocol for Repeated Clinical Examination and Critical Care Ultrasonography Within the Simple Intensive Care Studies
10:38

Observational Study Protocol for Repeated Clinical Examination and Critical Care Ultrasonography Within the Simple Intensive Care Studies

Published on: January 16, 2019

Testing Sensory and Multisensory Function in Children with Autism Spectrum Disorder
09:13

Testing Sensory and Multisensory Function in Children with Autism Spectrum Disorder

Published on: April 22, 2015

Related Experiment Videos

Last Updated: Jun 17, 2026

Functional Near-Infrared Spectroscopy Hyperscanning Study in Psychological Counseling
06:04

Functional Near-Infrared Spectroscopy Hyperscanning Study in Psychological Counseling

Published on: January 17, 2025

Observational Study Protocol for Repeated Clinical Examination and Critical Care Ultrasonography Within the Simple Intensive Care Studies
10:38

Observational Study Protocol for Repeated Clinical Examination and Critical Care Ultrasonography Within the Simple Intensive Care Studies

Published on: January 16, 2019

Testing Sensory and Multisensory Function in Children with Autism Spectrum Disorder
09:13

Testing Sensory and Multisensory Function in Children with Autism Spectrum Disorder

Published on: April 22, 2015

Main Results:

  • The model successfully explains the male predominance, age distribution, impact of sleep position changes, and seasonal variation in SIDS.
  • The model suggests distinct subsets within SIDS, potentially linked to respiratory infection and neurological factors.
  • A specific genetic hypothesis involving an X-linked allele protective against cerebral anoxia is proposed.

Conclusions:

  • The quadruple-risk model provides a comprehensive framework for understanding SIDS.
  • Confirmatory testing of the proposed X-linked genetic factor is essential.
  • Further research into genetic predispositions may elucidate SIDS mechanisms.