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Reversible postictal ataxic hemiparesis.

S K Bansal1, J S Chopra

  • 1Department of Neurology, Postgraduate Institute of Medical Education and Research, Chandigarh, India.

Italian Journal of Neurological Sciences
|February 1, 1991
PubMed
Summary
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Ataxic hemiparesis (AH), a condition with weakness and ataxia, can stem from cerebral cortex lesions. This study reveals AH as a rare postictal phenomenon, with patients recovering quickly.

Area of Science:

  • Neurology
  • Clinical Neuroscience

Background:

  • Ataxic hemiparesis (AH) is characterized by ipsilateral pyramidal weakness and motor ataxia.
  • Lesions in the pons or cerebro-cerebellar and corticospinal pathways can cause AH.
  • Differential diagnosis includes lobar ataxias, sensory ataxia, and spinal cord lesions.

Observation:

  • Three cases of AH caused by contralateral cerebral cortex lesions are presented.
  • Two cases were associated with postictal seizures.
  • One patient experienced left AH, focal motor seizures, and cortical memory deficits.

Findings:

  • Cerebral cortical lesions can manifest as ataxic hemiparesis.
  • Ataxic hemiparesis is reported as a novel postictal phenomenon.
  • All patients demonstrated rapid recovery within seven days.

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Implications:

  • This expands the understanding of clinical localization for ataxic hemiparesis.
  • Recognizing AH as a postictal event is crucial for accurate diagnosis.
  • The findings suggest transient cortical dysfunction can lead to AH symptoms.