Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Herniated Intervertebral Disc l: Introduction01:29

Herniated Intervertebral Disc l: Introduction

Intervertebral disc herniation refers to the displacement of the nucleus pulposus (the gel-like inner core of the disc) through a tear or weakened area in the annulus fibrosus (the outer fibrous ring). The displaced disc material extends beyond the normal boundaries of the disc space and may compress or irritate nearby spinal nerve roots or, less commonly, the spinal cord.Etiology and Risk FactorsHerniation commonly results from degeneration, in which aging reduces disc hydration and...
Spinal Cord Injury ll: Pathophysiology01:14

Spinal Cord Injury ll: Pathophysiology

Spinal cord injury progresses through two interconnected phases: primary injury and secondary injury.Primary InjuryPrimary injury happens at the moment of trauma and involves immediate mechanical damage to the spinal cord.Compression happens when broken vertebrae, herniated discs, or accumulating blood (such as a hematoma) press directly against the spinal cord, distorting its normal shape and function. In cases of contusion, the cord is bruised by a blunt force (like penetrating injuries or...
Secondary Spinal Cord Injury llI: Pathophysiology01:25

Secondary Spinal Cord Injury llI: Pathophysiology

Early Ischemia and Ionic ImbalanceWithin minutes of spinal cord injury, a secondary cascade begins, progressing over hours to weeks. Vascular damage reduces blood flow, causing ischemia and mitochondrial dysfunction. ATP depletion leads to ion pump failure, membrane depolarization, sodium influx, potassium efflux, and water accumulation, resulting in cellular swelling. Increased intracellular calcium further disrupts mitochondria and accelerates cellular injury.Excitotoxicity and Neuronal...

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Irreducible Dislocation of the Great Toe Interphalangeal Joint, Dorsal, and Plantar. Management and Long-term Evolution.

Journal of orthopaedic case reports·2023
Same author

Response to 'Prospective study of pediatric medial humeral epicondyle fractures nonoperatively treated. Clinical, radiologic, and functional evaluation at long term'.

Journal of pediatric orthopedics. Part B·2021
Same author

Prospective study of pediatric medial humeral epicondyle fractures nonoperatively treated. Clinical, radiologic, and functional evaluation at long term.

Journal of pediatric orthopedics. Part B·2020
Same author

Dupuytren's disease in a child: a long-term evolution after surgery.

Journal of pediatric orthopedics. Part B·2018
Same author

Study of fall risk-increasing drugs in elderly patients before and after a bone fracture.

Postgraduate medical journal·2017
Same author

Partial double-layered patella in a nondysplasic adolescent.

Journal of pediatric orthopedics. Part B·2016
Same journal

Mosaicplasty for the treatment of the osteochondral lesion in the femoral head.

Bulletin of the NYU hospital for joint diseases·2012
Same journal

Hibernoma--a case series with multimodality imaging and pathologic correlation.

Bulletin of the NYU hospital for joint diseases·2012
Same journal

Progressive migration of broken Kirschner wire into the proximal tibia following tension-band wiring technique of a patellar fracture--case report.

Bulletin of the NYU hospital for joint diseases·2012
Same journal

Multidrug-resistant Acinetobacter baumannii infection following para-articular steroid injection in the knee--a case report.

Bulletin of the NYU hospital for joint diseases·2012
Same journal

Extensor indicis proprius and extensor digitorum communis rupture after volar locked plating of the distal radius--a case report.

Bulletin of the NYU hospital for joint diseases·2012
Same journal

Bilateral four-part anterior fracture dislocation of the shoulder--a case report and review of literature.

Bulletin of the NYU hospital for joint diseases·2012
See all related articles

Related Experiment Video

Updated: Jun 14, 2026

Transtubular Endoscopic Posterolateral Decompression for L5-S1 Lumbar Lateral Disc Herniation
10:09

Transtubular Endoscopic Posterolateral Decompression for L5-S1 Lumbar Lateral Disc Herniation

Published on: October 14, 2022

L3-L4 dislocation without neurological lesions.

Angel M Hidalgo-Ovejero1, Serafín García-Mata, Francisco J Martínez-Lecea

  • 1Public University of Navarra, Pamplona, Spain. angelhidalgo@ya.com

Bulletin of the NYU Hospital for Joint Diseases
|March 30, 2010
PubMed
Summary
This summary is machine-generated.

This case report details an exceptional mid-lumbar vertebral dislocation (L3-L4) without neurological deficits, treated successfully with closed reduction and surgical stabilization. It highlights a rare injury pattern and effective management strategies for spinal trauma.

Related Experiment Videos

Last Updated: Jun 14, 2026

Transtubular Endoscopic Posterolateral Decompression for L5-S1 Lumbar Lateral Disc Herniation
10:09

Transtubular Endoscopic Posterolateral Decompression for L5-S1 Lumbar Lateral Disc Herniation

Published on: October 14, 2022

Area of Science:

  • Orthopedics
  • Neurosurgery
  • Trauma Surgery

Background:

  • Vertebral dislocations are severe injuries, more common at thoracolumbar and sacrolumbar junctions.
  • Mid-lumbar vertebral dislocations are exceptionally rare, with limited documented cases.

Observation:

  • A case of L3-L4 vertebral dislocation without neurological deficits is presented.
  • The injury resulted from a high-energy event, specifically an airplane crash.

Findings:

  • The patient had no initial neurological deficits.
  • Treatment involved closed reduction using a halo-bifemoral system, followed by surgical pedicle instrumentation and arthrodesis from L2 to L5.

Implications:

  • This case expands the understanding of rare mid-lumbar spinal injuries.
  • Successful management with closed reduction and surgical fusion demonstrates a viable treatment approach for such severe trauma.