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Silent pulmonary embolism in a patient with acute myocardial infarction and type B acute aortic dissection - a case
A Tomaszuk-Kazberuk1, B Sobkowicz, A Lewczuk
1Department of Cardiology, University Hospital in Bialystok, Bialystok, Poland. walkaz@poczta.fm
Insights
This case highlights the risk of pulmonary embolism in patients with aortic dissection. Anticoagulant therapy is crucial for bedridden patients, even with aortic dissection, to prevent life-threatening thrombosis.
Area of Science:
- Cardiology
- Vascular Surgery
- Pulmonary Medicine
Background:
- A 69-year-old male patient presented with type B aortic dissection and acute myocardial infarction.
- Initial conservative management was chosen, withholding prophylactic anticoagulation due to the aortic dissection.
- Thoracic stent-graft implantation was considered but not immediately performed.
Observation:
- An elective computed tomography (CT) scan unexpectedly revealed two large thrombi at the pulmonary trunk bifurcation.
- The patient was asymptomatic for pulmonary embolism despite the presence of significant thrombi.
- Immediate heparin therapy was initiated upon discovery of the thrombi.
Findings:
- The patient responded well to heparin therapy, with subsequent imaging showing no residual embolic material.
- Continuous oral anticoagulation was prescribed post-discharge.
- The case underscores the potential for undetected thrombosis in patients with aortic dissection.
Implications:
- This case raises critical questions regarding the necessity of prophylactic anticoagulation in bedridden patients with aortic dissection.
- Failure to administer anticoagulation prophylaxis could lead to severe clinical outcomes, including pulmonary embolism.
- The findings suggest a need for revised guidelines on anticoagulation strategies in complex cardiovascular conditions.
Abstract:
A 69-year-old man while being treated for type B aortic dissection was also found to have acute myocardial infarction. The patient initially was treated conservatively. Prophylactic anticoagulant treatment of potential thrombosis was not given because of aortic dissection. Stent-graft implantation to the thoracic aorta was considered at the time. Unexpectedly, elective computer tomography (CT), revealed 2 large thrombi at the bifurcation of the pulmonary trunk. The patient did not show any symptoms of pulmonary embolism. Heparin therapy was initiated immediately. The patient responded well to the therapy and on transesophageal echocardiography and subsequent CT no embolic material was found. Since that time, the patient is receiving oral anticoagulation. The case we present raises the question of anticoagulation prophylaxis in the presence of aortic dissection in bedridden patients. Failure to use such therapy in our patient could have resulted in an unfavorable clinical outcome.
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