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Related Concept Videos

Visual Agnosia01:12

Visual Agnosia

Visual agnosia is a condition characterized by the inability to recognize visually presented objects despite having normal vision. For instance, a person with visual agnosia can describe the shape and color of an object but cannot identify or name it. This impairment does not affect their visual field, acuity, color vision, brightness discrimination, language, or memory. An example of this condition in a social setting is someone at a dinner party asking for "that silver thing with a round end"...

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Ocular motor apraxia after sequential bilateral striatal infarctions.

Pil-Wook Chung1, Heui-Soo Moon, Hwa Suk Song

  • 1Department of Neurology, Kangbuk Samsung Hospital, Sungkyunkwan University School of Medicine, Seoul, Korea.

Journal of Clinical Neurology (Seoul, Korea)
|April 17, 2010
PubMed
Summary

Ocular motor apraxia, a condition affecting eye movements, can result from basal ganglia lesions, not just frontoparietal ones. This case highlights striatal infarctions as a potential cause of this rare disorder.

Keywords:
Basal gangliaMRIOcular motor apraxia

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Area of Science:

  • Neuroscience
  • Neurology
  • Ophthalmology

Background:

  • Ocular motor apraxia is typically associated with bilateral frontoparietal lesions.
  • The precise neuroanatomical correlates of ocular motor apraxia are not fully elucidated.

Purpose of the Study:

  • To report a case of ocular motor apraxia secondary to bilateral striatal infarctions.
  • To expand the understanding of the neuroanatomical basis of ocular motor apraxia.

Main Methods:

  • Case report detailing a patient with ocular motor apraxia.
  • Clinical assessment of eye movements, including saccades and smooth pursuits.
  • Magnetic Resonance Imaging (MRI) to identify brain lesions.

Main Results:

  • The patient exhibited impaired voluntary saccades and smooth pursuits in vertical and horizontal planes.
  • The vestibulo-ocular reflex remained intact.
  • MRI revealed an old left putaminal infarction and an acute right caudoputaminal infarction.

Conclusions:

  • Bilateral basal ganglia lesions, specifically striatal infarctions, can cause ocular motor apraxia.
  • This finding broadens the spectrum of brain regions implicated in ocular motor apraxia.