Search research articles

ABOUT JoVE

Overview Leadership Blog JoVE Help Center

AUTHORS

Publishing Process Editorial Board Scope & Policies Peer Review FAQ Submit

LIBRARIANS

Testimonials Subscriptions Access Resources Library Advisory Board FAQ

RESEARCH

JoVE Journal Methods Collections JoVE Encyclopedia of Experiments Archive

EDUCATION

JoVE Core JoVE Business JoVE Science Education JoVE Lab Manual Faculty Resource Center Faculty Site

Terms & Conditions of Use

Related Concept Videos

Glaucoma: Overview

Glaucoma: Overview

Glaucoma is an eye condition characterized by increased intraocular pressure that damages the retina and optic nerve, leading to irreversible blindness if left untreated. The human eye has various components, including the cornea, iris, pupil, lens, and optic nerve. Aqueous humor is secreted by the epithelium of the ciliary body in the posterior chamber and flows through the trabecular meshwork and canal of Schlemm, maintaining normal intraocular pressure. The trabecular meshwork and the canal...

Angle Closure Glaucoma: Treatment

Angle Closure Glaucoma: Treatment

Angle-closure glaucoma, or closed-angle glaucoma, is an eye condition where the iris bulges out and blocks the iridocorneal angle, resulting in a buildup of aqueous humor and increased intraocular pressure. Immediate medical attention is necessary due to the sudden onset of symptoms. The treatment for angle-closure glaucoma includes short-term and long-term approaches. Short-term treatment involves using eye drops like pilocarpine to lower intraocular pressure by increasing aqueous humor...

Accessory Structures of the Eye

Accessory Structures of the Eye

Optical perception, or vision, is an extraordinary sense dependent on converting light signals received via the ocular organs. These organs, known as eyes, are securely positioned within the bony cavities of the skull, called orbits. The orbits serve a dual purpose: a protective shield for the ocular globes and a stable attachment point for the soft ocular tissues. The eye's external protective mechanisms include the eyelids, which are edged with lashes that act as a barrier against foreign...

Open Angle Glaucoma: Treatment

Open Angle Glaucoma: Treatment

In open-angle glaucoma, the iridocorneal angle remains open, but the trabecular meshwork becomes stiff, slowing down the outflow of aqueous humor. This causes a buildup of aqueous humor in the anterior chamber, leading to a sudden increase in intraocular pressure. The treatment for open-angle glaucoma focuses on reducing the elevated intraocular pressure by either decreasing the secretion of aqueous humor or increasing its outflow.
Drugs such as carbonic anhydrase inhibitors, α2- and...

Photoreceptors and Visual Pathways

Photoreceptors and Visual Pathways

At the molecular level, visual signals trigger transformations in photopigment molecules, resulting in changes in the photoreceptor cell's membrane potential. The photon's energy level is denoted by its wavelength, with each specific wavelength of visible light associated with a distinct color. The spectral range of visible light, classified as electromagnetic radiation, spans from 380 to 720 nm. Electromagnetic radiation wavelengths exceeding 720 nm fall under the infrared category, whereas...

Muscles of the Eye

Muscles of the Eye

The muscles of the eye are sophisticated structures that control eye movement and focus, allowing for the precise and rapid adjustments necessary for vision. The human eye is controlled by ten muscles — six extraocular muscles, three intraocular muscles, and one primary eyelid retractor muscle.
Extraocular Muscles
The six extraocular muscles surround the eyeball and control its movements. They are responsible for a wide range of eye motions, including looking up, down, left, right, and rotating...

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by

Same author

[Hordeolum and Chalazion: Diagnosis and Therapy].

MMW Fortschritte der Medizin·2026

Same author

Comparison of Two Surgical Methods for the Treatment of Convergence Excess: Y-Split and Retro-equatorial Myopexy of the Medial Rectus Muscle.

Klinische Monatsblatter fur Augenheilkunde·2025

Same author

[Etiology of pediatric ocular trauma in a tertiary referral hospital in Tanzania].

Die Ophthalmologie·2025

Same author

Neonatal Sepsis Episodes and Retinopathy of Prematurity in Very Preterm Infants.

JAMA network open·2024

Same author

[Nontraumatic Orbital Haemorrhage into the Inferior Rectus Muscle].

Klinische Monatsblatter fur Augenheilkunde·2024

Same author

Quantitative Fundus Autofluorescence in the Developing and Maturing Healthy Eye.

Translational vision science & technology·2021

Search research articles

Related Experiment Video

Updated: Jun 12, 2026

A Surgical Approach for Optic Nerve Crush in a Rabbit Model

A Surgical Approach for Optic Nerve Crush in a Rabbit Model

Published on: July 8, 2025

Ocular fat embolism syndrome.

Martin M Nentwich¹, Matthias Remy, Ulrich C Schaller

¹Department of Ophthalmology, Ludwig-Maximilian University, Mathildenstr. 8, 80336, Munich, Germany. Martin.Nentwich@med.uni-muenchen.de

International Ophthalmology

|May 26, 2010

Summary

This summary is machine-generated.

Ocular fat embolism syndrome, a rare condition without heart defects, can affect the retina. This case highlights its occurrence in a young patient following a tibial fracture.

Related Experiment Videos

Last Updated: Jun 12, 2026

A Surgical Approach for Optic Nerve Crush in a Rabbit Model

A Surgical Approach for Optic Nerve Crush in a Rabbit Model

Published on: July 8, 2025

Area of Science:

Ophthalmology
Trauma Surgery
Medical Case Reports

Background:

Fat embolism syndrome (FES) is a serious complication, typically associated with long bone fractures.
Ocular involvement in FES is uncommon, and its occurrence without cardiac defects is exceptionally rare.
Ophthalmic manifestations of FES can include retinal changes, visual disturbances, and vision loss.

Observation:

A 16-year-old female presented with symptoms suggestive of FES after sustaining a tibial fracture.
The patient had no history or evidence of any cardiac defects.
Ophthalmic examination revealed retinal involvement consistent with fat embolism.

Findings:

The diagnosis of ocular fat embolism syndrome was confirmed in the absence of any cardiac abnormalities.
The retinal findings were attributed to the systemic fat embolism following the tibial fracture.
This case underscores the potential for ocular complications in FES, even in the absence of cardiac involvement.

Implications:

This case expands the understanding of the clinical spectrum of fat embolism syndrome.
Ophthalmologists should consider FES in the differential diagnosis of retinal abnormalities, particularly in patients with long bone fractures.
Further research is warranted to elucidate the mechanisms and management of ocular FES without cardiac defects.