Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Graves Disease II: Pathophysiology01:24

Graves Disease II: Pathophysiology

Graves’ disease is an autoimmune disorder characterized by the production of thyroid-stimulating immunoglobulins (TSI) that activate TSH receptors, leading to excessive synthesis and release of thyroid hormones (T3 and T4) and resulting in hyperthyroidism.Among all causes of hyperthyroidism, Graves’ disease is the most common and can happen at any age, though it is more frequent in women. It produces a hypermetabolic state with features such as weight loss, tachycardia, tremor, and heat...
Degenerative Disc Disease ll: Pathophysiology01:23

Degenerative Disc Disease ll: Pathophysiology

The symptoms of degenerative disc disease arise from a combination of mechanical compression, vascular compromise, and biochemical inflammation, which together disrupt nerve function and produce pain.Mechanical CompressionDisc degeneration reduces height and elasticity, predisposing to herniation of the nucleus pulposus, a major cause of radicular pain. Herniations may be protrusion (bulging with intact annulus), extrusion (nucleus extends beyond disc but remains connected), or sequestration...
Gastritis II: Pathophysiology01:26

Gastritis II: Pathophysiology

The pathophysiology of gastritis begins with the colonization of the stomach lining by Helicobacter pylori (H. pylori). This bacterium spreads mainly via the oral-oral route through saliva or shared utensils, and can also be transmitted in overcrowded or unhygienic environments through contaminated water, despite its brief survival outside the body.ColonizationOnce ingested, H. pylori enters the stomach and begins colonization by navigating through the mucus layer lining the stomach wall. It...
Gastritis-II: Pathophysiology01:17

Gastritis-II: Pathophysiology

Gastritis is marked by disruption of the mucosal barrier that usually protects the stomach tissue from digestive juices and manifests in acute and chronic forms.
In acute gastritis, the gastric mucosa becomes swollen and red and undergoes superficial erosion. Superficial ulceration may lead to bleeding.
In chronic gastritis, persistent or repeated insults lead to chronic inflammatory changes and, eventually, thinning or atrophy of the gastric tissue.
Gastritis can stem from various causes, each...
Secondary Spinal Cord Injury llI: Pathophysiology01:25

Secondary Spinal Cord Injury llI: Pathophysiology

Early Ischemia and Ionic ImbalanceWithin minutes of spinal cord injury, a secondary cascade begins, progressing over hours to weeks. Vascular damage reduces blood flow, causing ischemia and mitochondrial dysfunction. ATP depletion leads to ion pump failure, membrane depolarization, sodium influx, potassium efflux, and water accumulation, resulting in cellular swelling. Increased intracellular calcium further disrupts mitochondria and accelerates cellular injury.Excitotoxicity and Neuronal...
Cirrhosis II: Pathophysiology01:24

Cirrhosis II: Pathophysiology

Cirrhosis is a progressive chronic liver injury caused by prolonged inflammation, excessive fibrotic remodeling, and impaired regeneration. Over time, repeated hepatic insults disrupt the liver’s architecture and function, leading to reduced blood flow, impaired bile drainage, and diminished metabolic capacity.Pathophysiology of cirrhosisCirrhosis arises from three main responses to chronic liver damage: inflammation, immune activation, and hepatocyte death. These processes lead to structural...

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

[Colchicine in Cardiology Practice: Use in Atrial Fibrillation, Inflammatory Diseases, Heart Failure, and Cardiac Complications of COVID-19].

Kardiologiia·2026
Same author

[Colchicine in Cardiology Practice. Mechanisms of Influence on the Cardiovascular System, Use in the Treatment of Pericarditis and Ischemic Heart Disease].

Kardiologiia·2025
Same author

[From the history of the buildings of the Mandryka Central Military Clinical Hospital].

Voenno-meditsinskii zhurnal·2019
Same author

[SELFLESSNESS OF BESIEGED LENINGRAD: PROBLEMS OF MOTHERHOOD AND CHILDHOOD (ON THE OCCASION OF THE 72ND ANNIVERSARY OF LIFTING THE SIEGE)].

Klinicheskaia meditsina·2018
Same author

[REGRESSION OF ATHEROSCLECROTIC LESIONS: MEDICAMENTAL AND ALIMENTARY FACTORS].

Klinicheskaia meditsina·2018
Same author

[Boris Evgen'evich Votchal, an outstanding therapist and brilliant scientist, the founder of clinical pharmacology in Russia].

Klinicheskaia meditsina·2016
Same journal

[THE HISTORY OF DEPARTMENT OF FACULTY THERAPY, AT THE VORONEZH HIGHER EDUCATION INSTITUTION (ON THE OCCASION OF THE CENTENARY ANNIVERSARY OF N.N. BURDENKO VORONEZH STATE MEDICAL UNIVERSITY)].

Klinicheskaia meditsina·2018
Same journal

[KYOTO CONSENSUS - THE NEW ETIOLOGICAL CLASSIFICATION OF CHRONIC GASTRITIS AND ITS DISCUSSION].

Klinicheskaia meditsina·2018
Same journal

[PITUITARY ADENOMA APOPLEXY].

Klinicheskaia meditsina·2018
Same journal

[A CASE OF FAMILIAL PULMONARY AND HEPATIC ECHINOCOCCOSIS)].

Klinicheskaia meditsina·2018
Same journal

[SYSTEMIC LUPUS ERYTHEMATOSUS WITH LUPUS NEPHRITIS IN THE ELDERLY (CLINICAL OBSERVATION)].

Klinicheskaia meditsina·2018
Same journal

[TYPICAL ERRORS IN THERAPY OF GASTROESOPHAGEAL REFLUX DISEASE].

Klinicheskaia meditsina·2018
See all related articles

Related Experiment Video

Updated: Jun 11, 2026

Diagnosis and Surgical Treatment of Human Brucellar Spondylodiscitis
06:23

Diagnosis and Surgical Treatment of Human Brucellar Spondylodiscitis

Published on: May 23, 2021

[Spondylogenic visceropathies].

V B Simonenko, O V Davydov

    Klinicheskaia Meditsina
    |July 9, 2010
    PubMed
    Summary
    This summary is machine-generated.

    This study categorizes spondylogenic visceropathies into bronchial, cardiac, and abdominal vertebral syndromes based on clinical symptoms in 830 patients. It details their distinct features, diagnosis, and treatment approaches.

    More Related Videos

    Laparoscopic Splenectomy with Pericardial Devascularization for Hypersplenism and Esophageal Variceal Hemorrhage Due to Portal Hypertension
    04:00

    Laparoscopic Splenectomy with Pericardial Devascularization for Hypersplenism and Esophageal Variceal Hemorrhage Due to Portal Hypertension

    Published on: November 15, 2024

    Related Experiment Videos

    Last Updated: Jun 11, 2026

    Diagnosis and Surgical Treatment of Human Brucellar Spondylodiscitis
    06:23

    Diagnosis and Surgical Treatment of Human Brucellar Spondylodiscitis

    Published on: May 23, 2021

    Laparoscopic Splenectomy with Pericardial Devascularization for Hypersplenism and Esophageal Variceal Hemorrhage Due to Portal Hypertension
    04:00

    Laparoscopic Splenectomy with Pericardial Devascularization for Hypersplenism and Esophageal Variceal Hemorrhage Due to Portal Hypertension

    Published on: November 15, 2024

    Area of Science:

    • Orthopedics
    • Internal Medicine
    • Neurology

    Background:

    • Spondylogenic visceropathies represent a complex group of conditions.
    • Understanding their varied clinical presentations is crucial for effective management.
    • This study focuses on classifying these disorders based on affected organ systems.

    Observation:

    • A cohort of 830 patients with spondylogenic visceropathies was analyzed.
    • Patients were subgrouped based on predominant clinical symptoms: bronchial (n=80), cardiac (n=530), and abdominal (n=220) vertebral syndromes.
    • The study systematically documented the clinical manifestations within each subgroup.

    Findings:

    • Distinct clinical features were identified for bronchial, cardiac, and abdominal vertebral syndromes.
    • Differential diagnostic criteria were established to distinguish between these subtypes.
    • Effective treatment modalities for each syndrome were described.

    Implications:

    • This classification aids in precise diagnosis and targeted treatment of spondylogenic visceropathies.
    • Improved understanding of these vertebral syndromes can enhance patient outcomes.
    • The findings support a more specialized approach to managing these complex conditions.