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Updated: Jun 11, 2026

Nerve-sparing Mid-urethral Obstruction (NeMO) in Female Small Rodents

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Published on: April 25, 2017

The relationship between urethral mobility and parity.

K J Dickie¹, K L Shek, H P Dietz

¹Sydney Medical School Nepean, University of Sydney, Nepean Hospital, Penrith, New South Wales, Australia.

BJOG : an International Journal of Obstetrics and Gynaecology

|July 13, 2010

Summary

This summary is machine-generated.

Vaginal childbirth significantly increases urethral mobility, particularly after the first birth. This finding is crucial for understanding pelvic floor dysfunction and stress urinary incontinence in women.

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Anogenital Distance and Perineal Measurements of the Pelvic Organ Prolapse (POP) Quantification System

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Published on: September 20, 2018

Area of Science:

Urogynecology
Pelvic Floor Imaging
Female Lower Urinary Tract Symptoms

Background:

Urethral mobility is linked to stress urinary incontinence (SUI).
Mid-urethral mobility is particularly relevant.
Understanding factors influencing urethral mobility is essential for managing SUI and pelvic organ prolapse.

Purpose of the Study:

To investigate the relationship between segmental urethral mobility and vaginal parity.
To assess the impact of childbirth on urethral mobility in women undergoing urodynamic testing.

Main Methods:

Retrospective analysis of 3D translabial ultrasound data from 648 women.
Urethral mobility measured at rest and during Valsalva maneuver using a standardized coordinate system.
Urethral length divided into five segments to calculate mobility vectors.

Main Results:

Vaginal childbirth increased urethral mobility by approximately 20% across all segments (P < 0.009).
The first vaginal delivery had the most substantial impact, especially on mid-urethral mobility.
The distal urethra remained the least mobile segment irrespective of parity.

Conclusions:

A significant association exists between urethral mobility and vaginal delivery in women with pelvic floor dysfunction symptoms.
The first vaginal birth appears to be the primary driver of increased urethral mobility.
Findings contribute to understanding the biomechanics of pelvic floor dysfunction.