Related Experiment Videos
[Apical hypertrophic cardiomyopathy and coronary arteriovenous fistula]
M Penas Lado1, J Pasalodos, L Pérez Alvarez
1Servicio de Cardiología, Hospital Juan Canalejo, La Coruña.
Insights
This case report details a rare association between apical hypertrophic cardiomyopathy and coronary arteriovenous fistula in a male patient experiencing angina. Treatment with Verapamil effectively managed his symptoms, suggesting a potential shared developmental origin.
Area of Science:
- Cardiology
- Cardiovascular Medicine
- Medical Case Reports
Background:
- Apical hypertrophic cardiomyopathy (AHC) is a form of hypertrophic cardiomyopathy characterized by apical thickening.
- Coronary arteriovenous fistula (CAVF) is an abnormal connection between a coronary artery and a cardiac chamber or major vessel.
Observation:
- A 56-year-old male presented with anginal symptoms.
- The patient was diagnosed with both apical hypertrophic cardiomyopathy of the Japanese type and a coronary arteriovenous fistula.
Findings:
- Both AHC and CAVF can independently cause myocardial ischemia and angina.
- The co-occurrence of these conditions in the patient led to aggravated ischemic symptoms.
- Symptoms were successfully managed with Verapamil.
Implications:
- The rare coexistence of AHC and CAVF suggests a potential common etiological pathway, possibly a shared developmental error.
- This case highlights the importance of considering multiple rare cardiac abnormalities in patients with complex presentations.
- Further research may elucidate the underlying mechanisms linking these two distinct cardiovascular conditions.
Abstract:
We present a case of association of apical hypertrophic cardiomyopathy of the Japanese type and coronary arteriovenous fistula in a 56-year-old male who presented with anginal symptoms. Both cardiopathies can produce myocardial ischemia and angina, and their association could aggravate the ischemia. In our patient the symptoms were adequately controlled with Verapamil. The coexistence of these two rare entities in the same patient has recently been described in 2 other cases, allowing us to speculate on a possible etiological relation between the 2 abnormalities, probably both been originated in a common developmental error.