Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Pulmonary Embolism I: Introduction01:29

Pulmonary Embolism I: Introduction

Pulmonary embolism (PE) occurs when a thrombus, fat or air embolus, amniotic fluid, or tumor tissue blocks one or more pulmonary arteries. These blockages originate in the venous system or the right side of the heart.EtiologyPE primarily arises from deep vein thrombosis (DVT) and other hypercoagulable states, such as inherited thrombophilias. Additional etiological factors include venous stasis, commonly seen in obesity, and endothelial injury from surgery and trauma. Less common causes include...
Pulmonary Embolism I: Introduction01:19

Pulmonary Embolism I: Introduction

A blood clot, or thrombus, is a semi-solid mass composed of fibrin, platelets, and red blood cells. When it forms within a vessel, it can obstruct blood flow, known as thrombosis. If part of the clot detaches, it becomes an embolus that can travel and block distant vessels. When this occurs in the pulmonary arteries, it causes a condition known as pulmonary embolism (PE).Origin and ImpactMost often, the embolus originates from a thrombus in the deep veins of the lower limbs, a condition called...
Pulmonary Embolism III: Nursing Management01:27

Pulmonary Embolism III: Nursing Management

A pulmonary embolism occurs when a thrombus, amniotic fluid, tumor tissue, fat, or air embolus blocks one or more pulmonary arteries. Effective nursing management and patient education are crucial for improving outcomes and preventing recurrence.Nursing management starts with obtaining a comprehensive patient history, particularly noting any history of deep vein thrombosis (DVT). Assess for clinical manifestations, including dyspnea, chest pain, crackles, heart murmurs, and signs of right-sided...
Pulmonary Embolism II: Diagnostic Studies and Interprofessional Care01:29

Pulmonary Embolism II: Diagnostic Studies and Interprofessional Care

Diagnosing Pulmonary EmbolismDiagnosing pulmonary embolism (PE) involves clinical assessment and advanced imaging tests. The preferred diagnostic tool is the spiral (helical) CT scan or CT angiography (CTA), which uses intravenous contrast media to visualize the pulmonary vasculature and identify emboli.A ventilation-perfusion (V/Q) scan is an alternative for patients unable to receive contrast media. This scan includes both perfusion and ventilation scanning. Perfusion scanning involves...
Flail Chest-II01:26

Flail Chest-II

Managing flail chest, a condition characterized by a segment of the chest wall moving independently from the rest of the thoracic cage, requires a comprehensive approach. It includes a thorough assessment of the patient's condition, a diagnostic evaluation to determine the extent of the injury, and the implementation of appropriate medical interventions tailored to the individual's needs.
Assessment:
1. Clinical Evaluation:
History:
Pneumothorax-II01:27

Pneumothorax-II

Pneumothorax is a medical condition defined by the buildup of air in the pleural space between the lungs and the chest wall. This accumulation of air can lead to partial or complete lung collapse, resulting in a range of clinical manifestations. Understanding the clinical presentation and effective management strategies is crucial for healthcare professionals in providing timely and appropriate care to individuals with pneumothorax.
Clinical Manifestations:

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Inhibiting connexin channels protects against cryopreservation-induced cell death in human blood vessels.

European journal of vascular and endovascular surgery : the official journal of the European Society for Vascular Surgery·2013
Same author

Gap26, a connexin mimetic peptide, inhibits currents carried by connexin43 hemichannels and gap junction channels.

Pharmacological research·2012
Same author

Signalling of DNA damage and cytokines across cell barriers exposed to nanoparticles depends on barrier thickness.

Nature nanotechnology·2011
Same author

FULMINATING DYSENTERY IN A CHILD, CAUSED BY B. DYSENTERIAE SONNE.

British medical journal·2010
Same author

Polar nephelometer for atmospheric particulate studies.

Applied optics·2010
Same author

Differences in protein and glycoprotein patterns between mature and immature neutrophil leukocytes.

Biochimica et biophysica acta·2009

Related Experiment Video

Updated: Jun 9, 2026

A Porcine Model of Acute Autologous Pulmonary Embolism
07:44

A Porcine Model of Acute Autologous Pulmonary Embolism

Published on: September 6, 2024

A CASE OF EXTENSIVE PULMONARY EMBOLISM FOLLOWING FRACTURE

V Cotton-Cornwall, C W Ponder, W H Evans

    British Medical Journal
    |August 27, 2010
    PubMed
    Summary

    No abstract available in PubMed .

    More Related Videos

    Establishment of a Minimally Invasive Rat Model of Pulmonary Embolism Using Autologous Blood Clots
    08:02

    Establishment of a Minimally Invasive Rat Model of Pulmonary Embolism Using Autologous Blood Clots

    Published on: October 25, 2024

    Related Experiment Videos

    Last Updated: Jun 9, 2026

    A Porcine Model of Acute Autologous Pulmonary Embolism
    07:44

    A Porcine Model of Acute Autologous Pulmonary Embolism

    Published on: September 6, 2024

    Establishment of a Minimally Invasive Rat Model of Pulmonary Embolism Using Autologous Blood Clots
    08:02

    Establishment of a Minimally Invasive Rat Model of Pulmonary Embolism Using Autologous Blood Clots

    Published on: October 25, 2024