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Cyclosporin A suppresses cisplatin-induced oncogene expression in human cancer cells

K J Scanlon1, W Z Wang, H Han

  • 1Department of Medical Oncology, City of Hope National Medical Center, Duarte, CA 91010.

Cancer Treatment Reviews
|December 1, 1990
PubMed

Insights

Cyclosporin A reverses cisplatin resistance in ovarian cancer cells by suppressing DNA repair gene induction. This finding offers a potential strategy to enhance chemotherapy effectiveness against drug-resistant cancers.

Area of Science:

  • Molecular Biology
  • Cancer Research
  • Pharmacology

Background:

  • Chemotherapeutic agents often target DNA, and enhanced DNA repair is a key mechanism of cellular resistance.
  • Cisplatin-resistant human ovarian carcinoma cells (A2780DDP) exhibit increased gene expression for oncogenes and DNA repair enzymes upon treatment.
  • Cyclosporin A is known to reverse drug resistance, but its precise mechanism remains elusive.

Purpose of the Study:

  • To investigate the mechanism by which Cyclosporin A (CSA) reverses cisplatin resistance in ovarian cancer cells.
  • To determine the effect of CSA on the induction of genes involved in DNA repair pathways in resistant cells.

Main Methods:

  • A2780DDP cells, resistant to cisplatin, were exposed weekly to Cyclosporin A.
  • A revertant cell line (A2780DDP/CSA) that regained sensitivity to cisplatin was generated.
  • Gene expression analysis was performed to assess changes in oncogenes and DNA repair enzymes.

Main Results:

  • Weekly exposure to Cyclosporin A led to the development of a cisplatin-sensitive cell line (A2780DDP/CSA).
  • The revertant cell line (A2780DDP/CSA) showed suppressed induction of genes essential for repairing drug-induced DNA damage.
  • This suggests Cyclosporin A interferes with the cellular response mechanisms that confer cisplatin resistance.

Conclusions:

  • Cyclosporin A can re-sensitize cisplatin-resistant ovarian cancer cells to chemotherapy.
  • The mechanism involves the suppression of gene induction required for DNA repair.
  • This study highlights a potential therapeutic strategy for overcoming chemoresistance in ovarian cancer.

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