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Correlation of isolates from pyoderma and carrier sites.

A Chopra1, R Purl, R R Mittal

  • 1Department of Dermatology, Government Medical College, Patiala - 147001, India.

Indian Journal of Dermatology, Venereology and Leprology
|October 19, 2010
PubMed
Summary

A study of 100 pyoderma cases found a significant correlation between skin bacteria on lesions and normal skin or nasal flora. This suggests potential links between pyoderma and an individual's normal bacterial microbiome.

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Area of Science:

  • Dermatology
  • Microbiology
  • Clinical Science

Background:

  • Pyoderma is a common skin infection.
  • Understanding the source of pyoderma-causing bacteria is crucial for effective treatment.
  • The relationship between bacteria found on pyoderma lesions and those in normal skin or nasal flora requires further investigation.

Purpose of the Study:

  • To investigate the correlation between bacterial isolates from pyoderma lesions and those from normal skin and nasal flora in patients.
  • To identify the predominant microorganisms responsible for pyoderma and their antibiotic susceptibility.

Main Methods:

  • A study involving 100 patients diagnosed with pyoderma.
  • Collection of swabs from pyoderma lesions, healthy distant skin, and anterior nares for each patient.
  • Microscopic examination, bacterial culture, and antibiotic susceptibility testing were performed on collected samples.

Main Results:

  • Identical bacterial isolates were found between pyoderma lesions and normal skin/nasal flora in 37% of cases.
  • Staphylococcus aureus was the primary causative agent, identified in 35 out of 37 cases.
  • Antibiotic susceptibility patterns were consistent in 12 of the 37 cases with identical isolates.

Conclusions:

  • A significant correlation exists between bacteria found on pyoderma lesions and an individual's normal skin and nasal microbiome.
  • Staphylococcus aureus is the predominant pathogen in these pyoderma cases.
  • Findings suggest that the patient's own flora may play a role in the pathogenesis of pyoderma.